Relationship Between c-Jun N-terminal Kinase and Depression

被引：2

作者：

Ma, Hongpeng ^{[1
]}

机构：

[1] Shandong Univ, Sch Med, Jinan 250100, Peoples R China

来源：

2020 INTERNATIONAL CONFERENCE ON ENERGY, ENVIRONMENT AND BIOENGINEERING (ICEEB 2020) | 2020年 / 185卷

关键词：

EXTENDS LIFE-SPAN; PROTEIN-KINASE; JNK ACTIVATION; INSULIN-RESISTANCE; SIGNALING PATHWAY; OXIDATIVE STRESS; DOCKING SITE; CELL-DEATH; PHOSPHORYLATION; APOPTOSIS;

D O I：

10.1051/e3sconf/202018503029

中图分类号：

TE [石油、天然气工业]; TK [能源与动力工程];

学科分类号：

0807 ; 0820 ;

摘要：

Depression is one of the most common emotional disorders. The cause of depression is still not clear. C-Jun N-terminal kinase (JNK) is one number of mitogen activated protein kinase (MAPK) family, which is closely related to the occurrence of many diseases. At present, it is believed that JNK plays an important role in the parthenogenesis of depression, but the specific mechanism is not clear. This review will focus on the possible mechanism of JNK protein and JNK signaling pathway affecting the parthenogenesis of depression.

引用

页数：9

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