Metabolic alkalosis, bedside and bench

被引：13

作者：

Laski, Melvin E.

Sabatini, Sandra

机构：

[1] Texas Tech Univ, Hlth Sci Ctr, Dept Internal Med, Lubbock, TX 79413 USA

[2] Texas Tech Univ, Hlth Sci Ctr, Dept Physiol, Lubbock, TX 79413 USA

来源：

SEMINARS IN NEPHROLOGY | 2006年 / 26卷 / 06期

关键词：

metabolic alkalosis; acid-base regulation; bicarbonate transport; urinary acidification; endothelin; angiotensin; aldosterone;

D O I：

10.1016/j.semnephrol.2006.09.001

中图分类号：

R5 [内科学]; R69 [泌尿科学（泌尿生殖系疾病）];

学科分类号：

1002 ; 100201 ;

摘要：

Although significant contributions to the understanding of metabolic alkalosis have been made recently, much of our knowledge rests on data from clearance studies performed in humans and animals many years ago. This article reviews the contributions of these studies, as well as more recent work relating to the control of renal acid-base transport by mineralocorticoid hormones, angiotensin, endothelin, nitric oxide, and pottasium balance. Finally, clinical aspects of metabolic alkalosis are considered. © 2006 Elsevier Inc. All rights reserved.

引用

页码：404 / 421

页数：18

共 146 条

[1] P-31-NMR INVIVO MEASUREMENT OF RENAL INTRACELLULAR PH - EFFECTS OF ACIDOSIS AND K+ DEPLETION IN RATS [J].