Nitric oxide as a bifunctional regulator of apoptosis

被引：383
作者：
Kim, YM
Bombeck, CA
Billiar, TR
机构：
[1] Univ Pittsburgh, Dept Surg, Pittsburgh, PA USA
[2] Kangweon Natl Univ, Coll Med, Dept Cellular & Mol Biol, Chunchon, Kanwon Do, South Korea
来源：
CIRCULATION RESEARCH | 1999年 / 84卷 / 03期
关键词：
nitric oxide; apoptosis; caspase; cGMP; peroxynitrite;
D O I：
10.1161/01.RES.84.3.253
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：253 / 256
页数：4
相关论文
共 26 条
[1]  
ALBINA JE, 1993, J IMMUNOL, V150, P5080
[2]   Death receptors: Signaling and modulation [J].
Ashkenazi, A ;
Dixit, VM .
SCIENCE, 1998, 281 (5381) :1305-1308
[3]   THE NITRIC-OXIDE DONORS, AZIDE AND HYDROXYLAMINE, INHIBIT THE PROGRAMMED CELL-DEATH OF CYTOKINE-DEPRIVED HUMAN EOSINOPHILS [J].
BEAUVAIS, F ;
MICHEL, L ;
DUBERTRET, L .
FEBS LETTERS, 1995, 361 (2-3) :229-232
[4]   Suppression of apoptosis by nitric oxide via inhibition of interleukin-1 beta-converting enzyme (ICE)-like and cysteine protease protein (CPP)-32-like proteases [J].
Dimmeler, S ;
Haendeler, J ;
Nehls, M ;
Zeiher, AM .
JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 185 (04) :601-607
[5]  
FEHSEL K, 1995, J IMMUNOL, V155, P2858
[6]   Molecular biology of nitric oxide synthases [J].
Geller, DA ;
Billiar, TR .
CANCER AND METASTASIS REVIEWS, 1998, 17 (01) :7-23
[7]   SPLENIC B-LYMPHOCYTE PROGRAMMED CELL-DEATH IS PREVENTED BY NITRIC-OXIDE RELEASE THROUGH MECHANISMS INVOLVING SUSTAINED BCL-2 LEVELS [J].
GENARO, AM ;
HORTELANO, S ;
ALVAREZ, A ;
MARTINEZA, C ;
BOSCA, L .
JOURNAL OF CLINICAL INVESTIGATION, 1995, 95 (04) :1884-1890
[8]   Disruption of Fas receptor signaling by nitric oxide in eosinophils [J].
Hebestreit, H ;
Dibbert, B ;
Balatti, I ;
Braun, D ;
Schapowal, A ;
Blaser, K ;
Simon, HU .
JOURNAL OF EXPERIMENTAL MEDICINE, 1998, 187 (03) :415-425
[9]  
Heneka MT, 1998, J NEUROCHEM, V71, P88
[10]   MACROPHAGE CYTOTOXICITY - ROLE FOR L-ARGININE DEIMINASE AND IMINO-NITROGEN OXIDATION TO NITRITE [J].
HIBBS, JB ;
TAINTOR, RR ;
VAVRIN, Z .
SCIENCE, 1987, 235 (4787) :473-476
123