How do tumor cells respond to HDAC inhibition?

被引:93
作者
Newbold, Andrea [1 ,2 ]
Falkenberg, Katrina J. [3 ]
Prince, H. Miles [2 ,4 ]
Johnstone, Ricky W. [1 ,2 ]
机构
[1] Peter MacCallum Canc Ctr, Canc Therapeut Program, East Melbourne, Vic, Australia
[2] Univ Melbourne, Sir Peter MacCallum Dept Oncol, Parkville, Vic, Australia
[3] IMP Res Inst Mol Pathol, Vienna, Austria
[4] Peter MacCallum Canc Ctr, Div Canc Med, East Melbourne, Vic, Australia
基金
澳大利亚国家健康与医学研究理事会; 英国医学研究理事会;
关键词
apoptosis; cell cycle arrest; differentiation; histone deacetylase inhibitor; senescence; tumor immunogenicity; HISTONE DEACETYLASE INHIBITORS; ACID INDUCES DIFFERENTIATION; CANCER STEM-CELLS; DNA-DAMAGE; VALPROIC ACID; BREAST-CANCER; MYELOID-LEUKEMIA; MANTLE CELL; THERAPEUTIC ACTIVITIES; ANTICANCER ACTIVITIES;
D O I
10.1111/febs.13746
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
It is now well recognized that mutations, deregulated expression, and aberrant recruitment of epigenetic readers, writers, and erasers are fundamentally important processes in the onset and maintenance of many human tumors. The molecular, biological, and biochemical characteristics of a particular class of epigenetic erasers, the histone deacetylases (HDACs), have been extensively studied and small-molecule HDAC inhibitors (HDACis) have now been clinically approved for the treatment of human hemopoietic malignancies. This review explores our current understanding of the biological and molecular effects on tumor cells following HDACi treatment. The predominant responses include induction of tumor cell death and inhibition of proliferation that in experimental models have been linked to therapeutic efficacy. However, tumor cell-intrinsic responses to HDACi, including modulating tumor immunogenicity have also been described and may have substantial roles in mediating the antitumor effects of HDACi. We posit that the field has failed to fully reconcile the biological consequences of exposure to HDACis with the molecular events that underpin these responses, however progress is being made. Understanding the pleiotrophic activities of HDACis on tumor cells will hopefully fast track the development of more potent and selective HDACi that may be used alone or in combination to improve patient outcomes.
引用
收藏
页码:4032 / 4046
页数:15
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