No association between alpha 1-antichymotrypsin and familial Alzheimer's disease

被引：10
作者：
Haines, JL
Pritchard, ML
Saunders, AM
Schildkraut, JM
Growdon, JH
Gaskell, PC
Farrer, LA
Auerbach, SA
Gusella, JF
Locke, PA
Rosi, BL
Yamaoka, L
Small, GW
Conneally, PM
Roses, AD
PericakVance, M
机构：
[1] MASSACHUSETTS GEN HOSP,DEPT NEUROL,BOSTON,MA 02114
[2] DUKE UNIV,MED CTR,DUKE COMPREHENS CANC CTR,DURHAM,NC 27710
[3] BOSTON UNIV,MED CTR,DEPT NEUROL,BOSTON,MA 02118
[4] UNIV CALIF LOS ANGELES,SCH MED,DEPT PSYCHIAT & BIOBEHAV SCI,LOS ANGELES,CA 90024
[5] INDIANA UNIV,MED CTR,DEPT MED & MOL GENET,INDIANAPOLIS,IN 46202
来源：
APOLIPOPROTEIN E GENOTYPING IN ALZHEIMER'S DISEASE | 1996年 / 802卷
关键词：
D O I：
10.1111/j.1749-6632.1996.tb32596.x
中图分类号：
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号：
07 ; 0710 ; 09 ;
摘要：
引用
收藏
页码：35 / 41
页数：7
相关论文
共 21 条
[1]   ALPHA-1-ANTICHYMOTRYPSIN IS ASSOCIATED SOLELY WITH AMYLOID DEPOSITS CONTAINING THE BETA-PROTEIN - AMYLOID AND CELL LOCALIZATION OF ALPHA-1-ANTICHYMOTRYPSIN [J].
ABRAHAM, CR ;
SHIRAHAMA, T ;
POTTER, H .
NEUROBIOLOGY OF AGING, 1990, 11 (02) :123-129
[2]   IMMUNOCHEMICAL IDENTIFICATION OF THE SERINE PROTEASE INHIBITOR ALPHA-1-ANTICHYMOTRYPSIN IN THE BRAIN AMYLOID DEPOSITS OF ALZHEIMERS-DISEASE [J].
ABRAHAM, CR ;
SELKOE, DJ ;
POTTER, H .
CELL, 1988, 52 (04) :487-501
[3]   PROTECTIVE EFFECT OF APOLIPOPROTEIN-E TYPE-2 ALLELE FOR LATE-ONSET ALZHEIMER-DISEASE [J].
CORDER, EH ;
SAUNDERS, AM ;
RISCH, NJ ;
STRITTMATTER, WJ ;
SCHMECHEL, DE ;
GASKELL, PC ;
RIMMLER, JB ;
LOCKE, PA ;
CONNEALLY, PM ;
SCHMADER, KE ;
SMALL, GW ;
ROSES, AD ;
HAINES, JL ;
PERICAKVANCE, MA .
NATURE GENETICS, 1994, 7 (02) :180-184
[4]   GENE DOSE OF APOLIPOPROTEIN-E TYPE-4 ALLELE AND THE RISK OF ALZHEIMERS-DISEASE IN LATE-ONSET FAMILIES [J].
CORDER, EH ;
SAUNDERS, AM ;
STRITTMATTER, WJ ;
SCHMECHEL, DE ;
GASKELL, PC ;
SMALL, GW ;
ROSES, AD ;
HAINES, JL ;
PERICAKVANCE, MA .
SCIENCE, 1993, 261 (5123) :921-923
[5]   SEGREGATION OF A MISSENSE MUTATION IN THE AMYLOID PRECURSOR PROTEIN GENE WITH FAMILIAL ALZHEIMERS-DISEASE [J].
GOATE, A ;
CHARTIERHARLIN, MC ;
MULLAN, M ;
BROWN, J ;
CRAWFORD, F ;
FIDANI, L ;
GIUFFRA, L ;
HAYNES, A ;
IRVING, N ;
JAMES, L ;
MANT, R ;
NEWTON, P ;
ROOKE, K ;
ROQUES, P ;
TALBOT, C ;
PERICAKVANCE, M ;
ROSES, A ;
WILLIAMSON, R ;
ROSSOR, M ;
OWEN, M ;
HARDY, J .
NATURE, 1991, 349 (6311) :704-706
[6]   No genetic effect of alpha(1)-antichymotrypsin in Alzheimer disease [J].
Haines, JL ;
Pritchard, ML ;
Saunders, AM ;
Schildkraut, JM ;
Growdon, JH ;
Gaskell, PC ;
Farrer, LA ;
Auerbach, SA ;
Gusella, JF ;
Locke, PA ;
Rosi, BL ;
Yamaoka, L ;
Small, GW ;
Conneally, PM ;
Roses, AD ;
PericakVance, MA .
GENOMICS, 1996, 33 (01) :53-56
[7]  
HAINES JL, 1991, AM J HUM GENET, V48, P1021
[8]   APOE-ASTERISK-4-ASSOCIATED ALZHEIMERS-DISEASE RISK IS MODIFIED BY ALPHA-1-ANTICHYMOTRYPSIN POLYMORPHISM [J].
KAMBOH, MI ;
SANGHERA, DK ;
FERRELL, RE ;
DEKOSKY, ST .
NATURE GENETICS, 1995, 10 (04) :486-488
[9]  
LATHROP GM, 1985, AM J HUM GENET, V37, P482
[10]   CANDIDATE GENE FOR THE CHROMOSOME-1 FAMILIAL ALZHEIMERS-DISEASE LOCUS [J].
LEVYLAHAD, E ;
WASCO, W ;
POORKAJ, P ;
ROMANO, DM ;
OSHIMA, J ;
PETTINGELL, WH ;
YU, CE ;
JONDRO, PD ;
SCHMIDT, SD ;
WANG, K ;
CROWLEY, AC ;
FU, YH ;
GUENETTE, SY ;
GALAS, D ;
NEMENS, E ;
WIJSMAN, EM ;
BIRD, TD ;
SCHELLENBERG, GD ;
TANZI, RE .
SCIENCE, 1995, 269 (5226) :973-977
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