Mechanisms of GII.4 norovirus evolution

被引：136

作者：

Bull, Rowena A. ^{[1
,2
]}

White, Peter A. ^{[1
]}

机构：

[1] Univ New S Wales, Fac Sci, Sch Biotechnol & Biomol Sci, Sydney, NSW 2052, Australia

[2] Univ New S Wales, Fac Med, Sch Med Sci, Sydney, NSW 2052, Australia

来源：

TRENDS IN MICROBIOLOGY | 2011年 / 19卷 / 05期

关键词：

NORWALK-LIKE VIRUSES; BLOOD GROUP ANTIGEN; CAPSID PROTEIN FORMS; MOUTH-DISEASE VIRUS; HIGH MUTATION-RATES; INFLUENZA-A H3N2; UNITED-STATES; ACUTE GASTROENTERITIS; RNA VIRUSES; MOLECULAR CHARACTERIZATION;

D O I：

10.1016/j.tim.2011.01.002

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Since the late 1990 s norovirus (NoV) strains belonging to a single genotype (GII.4) have caused at least four global epidemics. To date, the higher epidemiological fitness of the GII.4 strains has been attributed to a faster rate of evolution within the virus capsid, resulting in the ability to escape herd immunity. Four key factors have been proposed to influence the rate of evolution in NoV. These include host receptor recognition, sequence space, duration of herd immunity, and replication kinetics. In this review we discuss recent advancements in our understanding of these four mechanisms in relation to GII.4 evolution.

引用

页码：233 / 240

页数：8

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