Living in context with the survival factor BAFF

被引：13

作者：

Cancro, Michael P. ^{[1
]}

机构：

[1] Univ Penn, Sch Med, Dept Pathol & Lab Med, Philadelphia, PA 19104 USA

来源：

IMMUNITY | 2008年 / 28卷 / 03期

关键词：

D O I：

10.1016/j.immuni.2008.02.010

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：300 / 301

页数：2

共 10 条

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[5] NF-κB1 p50 is required for BLyS attenuation of apoptosis but dispensable for processing of NF-κB2 p100 to p52 in quiescent mature B cells [J].

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[6] Transcriptional regulation via the NF-κB signaling module [J].

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[7] Reduced competitiveness of autoantigen-engaged B cells due to increased dependence on BAFF [J].

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