Design of Organo-Peptides As Bipartite PCSK9 Antagonists

被引:15
作者
Burdick, Daniel J. [2 ]
Skelton, Nicholas J. [2 ]
Ultsch, Mark [3 ]
Beresini, Maureen H. [4 ]
Eigenbrot, Charles [3 ]
Li, Wei [1 ]
Zhang, Yingnan [1 ]
Huy Nguyen [2 ]
Kong-Beltran, Monica [5 ]
Quinn, John G. [4 ]
Kirchhofer, Daniel [1 ]
机构
[1] Genentech Inc, Dept Early Discovery Biochem, San Francisco, CA 94080 USA
[2] Genentech Inc, Dept Discovery Chem, San Francisco, CA 94080 USA
[3] Genentech Inc, Dept Struct Biol, San Francisco, CA 94080 USA
[4] Genentech Inc, Dept Biochem & Cellular Pharmacol, San Francisco, CA 94080 USA
[5] Genentech Inc, Dept Mol Biol, San Francisco, CA 94080 USA
基金
美国国家卫生研究院;
关键词
DENSITY-LIPOPROTEIN RECEPTOR; SECRETED PCSK9; LDL; PROTEIN; BINDING; DISCOVERY; ANTIBODIES; SITE;
D O I
10.1021/acschembio.9b00899
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Proprotein convertase subtilisin/kexin 9 (PCSK9) has become an important therapeutic target for lipid lowering, since it regulates low-density lipoprotein cholesterol (LDL-c) levels by binding to liver LDL receptors (LDLR) and effecting their intracellular degradation. However, the development of small molecule inhibitors is hampered by the lack of attractive PCSK9 target sites. We recently discovered helical peptides that are able to bind to a cryptic groove site on PCSK9, which is situated in proximity to the main LDLR binding site. Here, we designed potent bipartite PCSK9 inhibitors by appending organic moieties to a helical groove-binding peptide to reach a hydrophobic pocket in the proximal LDLR binding region. The ultimately designed 1-amino-4-phenylcyclohexane-1-carbonyl extension improved the peptide affinity by >100-fold, yielding organo-peptide antagonists that potently inhibited PCSK9 binding to LDLR and preserved cellular LDLR. These new bipartite antagonists have reduced mass and improved potency compared to the first-generation peptide antagonists, further validating the PCSK9 groove as a viable therapeutic target site.
引用
收藏
页码:425 / 436
页数:12
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