The gasotransmitter hydrogen sulfide in hemodialysis patients

被引：1

作者：

Perna, Alessandra F. ^{[1
]}

Sepe, Immacolata ^{[1
]}

Lanza, Diana ^{[1
]}

Capasso, Rosanna ^{[2
]}

Di Marino, Veronica ^{[1
]}

De Santo, Natale G. ^{[1
]}

Ingrosso, Diego ^{[2
]}

机构：

[1] Second Univ Med, Dept Clin & Expt Internal Med, Div Nephrol 1, IT-80131 Naples, Italy

[2] Second Univ Med, Dept Biochem & Biophys F Cedrangolo, IT-80131 Naples, Italy

来源：

JOURNAL OF NEPHROLOGY | 2010年 / 23卷

关键词：

Cystathionine gamma-lyase; Hemodialysis; Hydrogen sulfide; Sulfidric acid; CARDIOPROTECTIVE ROLE; H2S; UREMIA; HYPERHOMOCYSTEINEMIA; ACCUMULATION; VASORELAXANT; HYPERTENSION; METHYLATION; FAILURE;

D O I：

暂无

中图分类号：

R5 [内科学]; R69 [泌尿科学（泌尿生殖系疾病）];

学科分类号：

1002 ; 100201 ;

摘要：

Hydrogen sulfide, H2S, is the third endogenous gas with cardiovascular properties (the others are nitric oxide and carbon monoxide). In fact, among other important signaling functions, H2S plays a key role in regulating blood pressure. Cystathionine beta-synthase, cystathionine gamma-lyase (CSE) and 3-mercaptopyruvate sulfurtransferase are the principal enzymes devoted to H2S formation. We have recently shown that H2S levels are decreased in patients on chronic hemodialysis through the transcriptional deregulation of the CSE gene, hinting at the possibility that a link exists between this finding and hypertension and the high cardiovascular mortality typical of these patients.

引用

页码：S92 / S96

页数：5

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