Kinetics of BCR-ABL after TKI Interruption during Pregnancy in CML: A Multinational Retrospective Analysis

被引：4
|
作者：
Lee, Jeong-Ok [1 ]
Kim, Dong-Wook [2 ]
Abruzzese, Elisabetta [3 ]
Apperley, Jane [4 ]
Caldwell, Louise [5 ]
Mauro, Michael J. [6 ]
机构：
[1] Seoul Natl Univ, Dept Internal Med, Bundang Hosp, Seongnam, South Korea
[2] Catholic Univ Korea, Seoul St Marys Hosp, Catholic Blood & Marrow Transplantat Ctr, Leukemia Res Inst,Dept Hematol,Coll Med, Seoul, South Korea
[3] S Eugenio Hosp, Haematol Unit, Rome, Italy
[4] Imperial Coll London, Ctr Haematol, London, England
[5] Univ Birmingham, Med Sch, Sheffield, S Yorkshire, England
[6] Mem Sloan Kettering Canc Ctr, Myeloproliferat Neoplasms Program, Leukemia Serv, 1275 York Ave, New York, NY 10021 USA
来源：
BLOOD | 2018年 / 132卷
关键词：
D O I：
10.1182/blood-2018-99-119615
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
4263
引用
收藏
页数：4
相关论文
共 50 条
  • [1] Kinetics of BCR-ABL Mutant Clones Determines Resistance in CML on Second Generation TKI Treatment
    Gruber, Franz X.
    Ernst, Thomas
    Maier, Jaqueline
    Mueller, Martin C.
    Mikkola, Ingvild
    Porkka, Kimmo
    Niederwieser, Dietger
    Hochhaus, Andreas
    Lange, Thoralf
    BLOOD, 2008, 112 (11) : 741 - 741
  • [2] BCR-ABL mutant kinetics in CML patients treated with dasatinib
    Nicolini, Franck E.
    Chabane, Kaddour
    Tigaud, Isabelle
    Michallet, Mauricette
    Magaud, Jean-Pierre
    Hayette, Sandrine
    LEUKEMIA RESEARCH, 2007, 31 (06) : 865 - 868
  • [3] Mutagenic analysis of bcr-abl in induction of CML in mice.
    Ren, RB
    Zhang, XW
    BLOOD, 1997, 90 (10) : 871 - 871
  • [4] Analysis of mutations in the BCR-ABL TK domain in CML patients
    Nesslboeck, M.
    Preuner, S.
    Lion, T.
    KLINISCHE PADIATRIE, 2010, 222 (03): : 217 - 217
  • [5] AUTOPHAGY GENES SUPPRESSED IN CML ARE UP-REGULATED FOLLOWING BCR-ABL INHIBITION WITH TKI
    Mukhopadhyay, A.
    Sahasrabudhe, N.
    Karvela, M.
    Allan, E.
    Chan, E.
    Helgason, G.
    Holyoake, T.
    HAEMATOLOGICA, 2012, 97 : 67 - 67
  • [6] Proliferation Kinetics of Subclones Carrying Point Mutations In the BCR-ABL TKD During TKI Treatment In CML Patients: Quantitative Monitoring by LD-PCR.
    Preuner, Sandra
    Bernroitner, Margit
    Mitterbauer, Gerlinde
    Mannhalter, Christine
    Herndlhofer, Susanne
    Sperr, Wolfgang R.
    Webersinke, Gerald
    Valent, Peter
    Lion, Thomas
    BLOOD, 2010, 116 (21) : 933 - 934
  • [7] PROGNOSIS FOR CML PATIENTS WITH >10% BCR-ABL AFTER 3 MONTHS OF IMATINIB DEPENDS ON THE INITIAL RATE OF BCR-ABL DECLINE
    Branford, S.
    Yeung, D.
    Parker, W. T.
    Purins, L.
    Braley, J.
    Seymour, J. F.
    Ross, D. M.
    Roberts, N. D.
    Hughes, T. P.
    HAEMATOLOGICA, 2014, 99 : 74 - 74
  • [8] BCR-ABL EXPRESSION IS REDUCED IN THE PRIMITIVE PRECURSOR FRACTION FROM CML TKI-TREATED PATIENTS
    Ruiz, M. S.
    Trasci, S. Baffa
    Moiraghi, B.
    Perez, M.
    Burgos, R.
    Foncuberta, C.
    Vitriu, A.
    Pavlovsky, C.
    Mordoh, J.
    Larripa, I.
    Sanchez Avalos, J. C.
    Bianchini, M.
    HAEMATOLOGICA, 2015, 100 : 429 - 429
  • [9] A Low Concentration of ABL001 Potentiates In Vitro TKI-Induced Bcr-Abl Kinase Inhibition in CML Cells
    Saunders, Verity A.
    Wang, Jueqiong
    Lu, Liu
    Eadie, Laura N.
    McLean, Jennifer A.
    Goyne, Jarrad M.
    Yeung, David T.
    White, Deborah L.
    Hughes, Timothy P.
    BLOOD, 2016, 128 (22)
  • [10] Reconstitution with Bcr-Abl negative recipient haematopoiesis after allogeneic BMT for CML
    Serrano, J
    Román, J
    Castillejo, JA
    Navarro, JA
    Pascual, A
    García-Rubio, I
    Falcón, M
    Martín, C
    Sánchez, J
    Herrera, C
    Torres, A
    BRITISH JOURNAL OF HAEMATOLOGY, 1998, 102 (01) : 318 - 318
12345