Defective dopamine receptor G-protein function contributes to reduced dopamine-mediated inhibition of Na,K-ATPase in proximal tubules of obese Zucker rats.

被引：0
|
作者：
Hussain, T [1 ]
Beheray, SA [1 ]
Lokhandwala, MF [1 ]
机构：
[1] Univ Houston, Coll Pharm, Inst Cardiovasc Studies, Houston, TX 77030 USA
来源：
AMERICAN JOURNAL OF HYPERTENSION | 1999年 / 12卷 / 04期
关键词：
dopamine; Na; K-ATPase; kidney; obesity; hypertension;
D O I：
暂无
中图分类号：
R6 [外科学];
学科分类号：
1002 ; 100210 ;
摘要：
引用
收藏
页码：68A / 68A
页数：1
相关论文
共 22 条
  • [21] Loss of NHERF-1 expression prevents dopamine-mediated Na-K-ATPase regulation in renal proximal tubule cells from rat models of hypertension: aged F344 rats and spontaneously hypertensive rats
    Barati, Michelle T.
    Ketchem, Corey J.
    Merchant, Michael L.
    Kusiak, Walter B.
    Jose, Pedro A.
    Weinman, Edward J.
    LeBlanc, Amanda J.
    Lederer, Eleanor D.
    Khundmiri, Syed J.
    AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY, 2017, 313 (02): : C197 - C206
  • [22] Tempol reduces oxidative stress, improves insulin sensitivity, decreases renal dopamine D1 receptor hyperphosphorylation, and restores D1 receptor-G-protein coupling and function in obese Zucker rats
    Banday, AA
    Marwaha, A
    Tallam, LS
    Lokhandwala, MF
    DIABETES, 2005, 54 (07) : 2219 - 2226
123