Regulation of bone mass in endocrine diseases including diabetes

被引：6
作者：
Higham, Claire [1 ,2 ]
Abrahamsen, Bo [3 ,4 ,5 ]
机构：
[1] Christie Hosp NHS Fdn Trust, Manchester, Lancs, England
[2] Univ Manchester, Manchester, Lancs, England
[3] Univ Southern Denmark, Dept Clin Res, Open Patient Data Exploratory Network, Odense, Denmark
[4] Holbaek Cent Hosp, Dept Med, Holbaek, Denmark
[5] Univ Oxford, NDORMS, Oxford, England
来源：
BEST PRACTICE & RESEARCH CLINICAL ENDOCRINOLOGY & METABOLISM | 2022年 / 36卷 / 02期
关键词：
thyrotoxicosis; primary hyperparathyroidism; type 1 and type 2 diabetes mellitus; Cushing's syndrome; growth hormone; low bone mass; GROWTH-HORMONE DEFICIENCY; MAJOR OSTEOPOROTIC FRACTURES; MINERAL DENSITY; POSTMENOPAUSAL WOMEN; ADULT-ONSET; HYPOPITUITARY PATIENTS; REPLACEMENT THERAPY; VERTEBRAL FRACTURES; CORTICOSTEROID USE; RISK;
D O I：
10.1016/j.beem.2022.101614
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
Hormonal regulation plays a key role in determining bone mass in humans. Both skeletal growth and bone loss in health and disease is critically controlled by endocrine factors and low bone mass is a feature of both excess and deficiency of a broad range of hormones. This article explores the impact of diabetes and thyroid, parathyroid, sex steroid and growth hormone disorders on bone mass and fracture risk. Evidence for current management strategies is provided along with suggested practice points and gaps in knowledge for future research.(c) 2022 The Author(s). Published by Elsevier Ltd. This is an open access article under the CC BY-NC-ND license (http:// creativecommons.org/licenses/by-nc-nd/4.0/).
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页数：14
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