Protein kinase C-α inhibits the repair of oxidative phosphorylation after S-(1,2-dichlorovinyl)-L-cysteine injury in renal proximal tubular cells.

被引：0

作者：

Nowak, G ^{[1
]}

Liu, XL ^{[1
]}

Godwin, ML ^{[1
]}

机构：

[1] Univ Arkansas Med Sci, Dept Pharmaceut Sci, Little Rock, AR 72205 USA

来源：

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY | 2003年 / 14卷

关键词：

D O I：

暂无

中图分类号：

R5 [内科学]; R69 [泌尿科学（泌尿生殖系疾病）];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：568A / 568A

页数：1

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[1] Protein kinase C-α inhibits the repair of oxidative phosphorylation after S-(1,2-dichlorovinyl)-L-cysteine injury in renal cells
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[4] Modulation of gene expression in human proximal tubular (hPT) cells by S-(1,2-dichlorovinyl)-L-cysteine (DCVC).
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[7] TRANSPORT AND ACTIVATION OF S-(1,2-DICHLOROVINYL)-L-CYSTEINE AND N-ACETYL-S-(1,2-DICHLOROVINYL)-L-CYSTEINE IN RAT-KIDNEY PROXIMAL TUBULES
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[8] S-(1,2-DICHLOROVINYL)-L-HOMOCYSTEINE (DCVHC), AN ANALOG OF THE RENAL TOXIN S-(1,2-DICHLOROVINYL)-L-CYSTEINE (DCVC), IS A POTENT NEPHROTOXIN
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[10] N-ACETYL S-(1,2-DICHLOROVINYL)-L-CYSTEINE PRODUCES A SIMILAR TOXICITY TO S-(1,2-DICHLOROVINYL)-L-CYSTEINE IN RABBIT RENAL SLICES - DIFFERENTIAL TRANSPORT AND METABOLISM
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