Dementia with lewy bodies

[31] KOSAKA K, 1993, ADV NEUROL, V60, P456

[32] KOSAKA K, 1978, ACTA NEUROPATHOL, V42, P127, DOI 10.1007/BF00690978

[33] KOSAKA K, 1984, CLIN NEUROPATHOL, V3, P185

[34] Kosaka K., 1980, PSYCHIAT NEUROL JAPO, V82, P292

[35] LEWY BODIES IN THE BRAIN OF 2 MEMBERS OF A FAMILY WITH THE 717 (VAL TO ILE) MUTATION OF THE AMYLOID PRECURSOR PROTEIN GENE [J]. NEUROSCIENCE LETTERS, 1994, 172 (1-2) : 77 - 79

[36] ANTI-UBIQUITIN IMMUNOCYTOCHEMISTRY IS MORE SENSITIVE THAN CONVENTIONAL TECHNIQUES IN THE DETECTION OF DIFFUSE LEWY BODY DISEASE [J]. JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY, 1989, 52 (01) : 67 - 71

[37] Lewy bodies contain altered α-synuclein in brains of many familiar Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes [J]. AMERICAN JOURNAL OF PATHOLOGY, 1998, 153 (05) : 1365 - 1370

[38] Lippa CF, 1999, ANN NEUROL, V45, P353, DOI 10.1002/1531-8249(199903)45:3<353::AID-ANA11>3.0.CO

[39] 2-4

[40] UBIQUITIN IS A COMMON FACTOR IN INTERMEDIATE FILAMENT INCLUSION-BODIES OF DIVERSE TYPE IN MAN, INCLUDING THOSE OF PARKINSONS-DISEASE, PICKS DISEASE, AND ALZHEIMERS-DISEASE, AS WELL AS ROSENTHAL FIBERS IN CEREBELLAR ASTROCYTOMAS, CYTOPLASMIC BODIES IN MUSCLE, AND MALLORY BODIES IN ALCOHOLIC LIVER-DISEASE [J]. JOURNAL OF PATHOLOGY, 1988, 155 (01) : 9 - 15

[31] KOSAKA K, 1993, ADV NEUROL, V60, P456

[32] KOSAKA K, 1978, ACTA NEUROPATHOL, V42, P127, DOI 10.1007/BF00690978

[33] KOSAKA K, 1984, CLIN NEUROPATHOL, V3, P185

[34] Kosaka K., 1980, PSYCHIAT NEUROL JAPO, V82, P292

[35] LEWY BODIES IN THE BRAIN OF 2 MEMBERS OF A FAMILY WITH THE 717 (VAL TO ILE) MUTATION OF THE AMYLOID PRECURSOR PROTEIN GENE [J]. NEUROSCIENCE LETTERS, 1994, 172 (1-2) : 77 - 79

[36] ANTI-UBIQUITIN IMMUNOCYTOCHEMISTRY IS MORE SENSITIVE THAN CONVENTIONAL TECHNIQUES IN THE DETECTION OF DIFFUSE LEWY BODY DISEASE [J]. JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY, 1989, 52 (01) : 67 - 71

[37] Lewy bodies contain altered α-synuclein in brains of many familiar Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes [J]. AMERICAN JOURNAL OF PATHOLOGY, 1998, 153 (05) : 1365 - 1370

[38] Lippa CF, 1999, ANN NEUROL, V45, P353, DOI 10.1002/1531-8249(199903)45:3<353::AID-ANA11>3.0.CO

[39] 2-4

[40] UBIQUITIN IS A COMMON FACTOR IN INTERMEDIATE FILAMENT INCLUSION-BODIES OF DIVERSE TYPE IN MAN, INCLUDING THOSE OF PARKINSONS-DISEASE, PICKS DISEASE, AND ALZHEIMERS-DISEASE, AS WELL AS ROSENTHAL FIBERS IN CEREBELLAR ASTROCYTOMAS, CYTOPLASMIC BODIES IN MUSCLE, AND MALLORY BODIES IN ALCOHOLIC LIVER-DISEASE [J]. JOURNAL OF PATHOLOGY, 1988, 155 (01) : 9 - 15