From neutrophil extracellular traps release to thrombosis: an overshooting host-defense mechanism?

被引：48

作者：

Borissoff, J. I.

Ten Cate, H. ^{[1
,2
]}

机构：

[1] Maastricht Univ Med Ctr, Lab Clin Thrombosis & Hemostasis, Dept Internal Med, Cardiovasc Res Inst Maastricht, NL-6200 MD Maastricht, Netherlands

[2] Maastricht Univ Med Ctr, Lab Clin Thrombosis & Hemostasis, Dept Biochem, Cardiovasc Res Inst Maastricht, NL-6200 MD Maastricht, Netherlands

来源：

JOURNAL OF THROMBOSIS AND HAEMOSTASIS | 2011年 / 9卷 / 09期

关键词：

ACTIVATED PROTEIN-C; CATHEPSIN-G; EXPRESSION; HISTONES; INACTIVATION; INFLAMMATION; MEDIATORS; PLATELETS; ELASTASE; PROMOTES;

D O I：

10.1111/j.1538-7836.2011.04425.x

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1791 / 1794

页数：4

共 42 条

[1] Extracellular histones increase plasma thrombin generation by impairing thrombomodulin-dependent protein C activation [J].

Ammollo, C. T. ;

Semeraro, F. ;

Xu, J. ;

Esmon, N. L. ;

Esmon, C. T. .

JOURNAL OF THROMBOSIS AND HAEMOSTASIS, 2011, 9 (09) :1795-1803

[2]

[Anonymous], COCHRANE DATABASE SY

[3] Role of polymorphonuclear neutrophils in atherosclerosis: Current state and future perspectives [J].

Baetta, Roberta ;

Corsini, Alberto .

ATHEROSCLEROSIS, 2010, 210 (01) :1-13

[4] MECHANISMS OF DISEASE The Hemostatic System as a Modulator of Atherosclerosis [J].

Borissoff, Julian Ilcheff ;

Spronk, Henri M. H. ;

ten Cate, Hugo .

NEW ENGLAND JOURNAL OF MEDICINE, 2011, 364 (18) :1746-1760

[5] Early Atherosclerosis Exhibits an Enhanced Procoagulant State [J].

Borissoff, Julian Ilcheff ;

Heeneman, Sylvia ;

Kilinc, Evren ;

Kassak, Peter ;

Van Oerle, Rene ;

Winckers, Kristien ;

Govers-Riemslag, Jose W. P. ;

Hamulyak, Karly ;

Hackeng, Tilman M. ;

Daemen, Mat J. A. P. ;

Ten Cate, Hugo ;

Spronk, Henri M. H. .

CIRCULATION, 2010, 122 (08) :821-U145

[6] Neutrophil extracellular traps kill bacteria [J].

Brinkmann, V ;

Reichard, U ;

Goosmann, C ;

Fauler, B ;

Uhlemann, Y ;

Weiss, DS ;

Weinrauch, Y ;

Zychlinsky, A .

SCIENCE, 2004, 303 (5663) :1532-1535

[7]

BROWER MS, 1982, J BIOL CHEM, V257, P9849

[8] Neutrophils in development of multiple organ failure in sepsis [J].

Brown, K. A. ;

Brain, S. D. ;

D Pearson, J. ;

D Edgeworth, J. ;

Lewis, S. M. ;

Treacher, D. F. .

LANCET, 2006, 368 (9530) :157-169

[9] Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood [J].

Clark, Stephen R. ;

Ma, Adrienne C. ;

Tavener, Samantha A. ;

McDonald, Braedon ;

Goodarzi, Zahra ;

Kelly, Margaret M. ;

Patel, Kamala D. ;

Chakrabarti, Subhadeep ;

McAvoy, Erin ;

Sinclair, Gary D. ;

Keys, Elizabeth M. ;

Allen-Vercoe, Emma ;

DeVinney, Rebekah ;

Doig, Christopher J. ;

Green, Francis H. Y. ;

Kubes, Paul .

NATURE MEDICINE, 2007, 13 (04) :463-469

[10] The lectin-like domain of thrombomodulin confers protection from neutrophil-mediated tissue damage by suppressing adhesion molecule expression via nuclear factor κB and mitogen-activated protein kinase pathways [J].

Conway, EM ;

Van de Wouwer, M ;

Pollefeyt, S ;

Jurk, K ;

Van Aken, H ;

De Vriese, A ;

Weitz, JI ;

Weiler, H ;

Hellings, PW ;

Schaeffer, P ;

Herbert, JM ;

Collen, D ;

Theilmeier, G .

JOURNAL OF EXPERIMENTAL MEDICINE, 2002, 196 (05) :565-577

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