The termination of PI3K signalling by SHIP1 and SHIP2 inositol 5-phosphatases

被引：74
|
作者：
Backers, K [1 ]
Blero, D [1 ]
Paternotte, N [1 ]
Zhang, J [1 ]
Erneux, C [1 ]
机构：
[1] Free Univ Brussels, Interdisciplinary Res Inst IRIBHM, B-1070 Brussels, Belgium
来源：
ADVANCES IN ENZYME REGULATION, VOL 43 | 2003年 / 43卷
关键词：
D O I：
10.1016/S0065-2571(02)00043-2
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
引用
收藏
页码：15 / 28
页数：14
相关论文
共 50 条
  • [21] Preclinical Evaluation of a Novel SHIP1 Phosphatase Activator for Inhibition of PI3K Signaling in Malignant B Cells
    Lemm, Elizabeth A.
    Valle-Argos, Beatriz
    Smith, Lindsay D.
    Richter, Johanna
    Gebreselassie, Yohannes
    Carter, Matthew J.
    Karolova, Jana
    Svaton, Michael
    Helman, Karel
    Weston-Bell, Nicola J.
    Karydis, Laura
    Williamson, Chris T.
    Lenz, Georg
    Pettigrew, Jeremy
    Harwig, Curtis
    Stevenson, Freda K.
    Cragg, Mark
    Forconi, Francesco
    Steele, Andrew J.
    Cross, Jennifer
    Mackenzie, Lloyd
    Klener, Pavel
    Packham, Graham
    CLINICAL CANCER RESEARCH, 2020, 26 (07) : 1700 - 1711
  • [22] TREM2-and DAP12-Dependent Activation of PI3K Requires DAP10 and Is Inhibited by SHIP1
    Peng, Qisheng
    Malhotra, Shikha
    Torchia, James A.
    Kerr, William G.
    Coggeshall, K. Mark
    Humphrey, Mary Beth
    SCIENCE SIGNALING, 2010, 3 (122) : ra38
  • [23] 5′-inositol phosphatase 2 (SHIP2) is over-expressed in endometrial cancer
    Sever, M
    Tandon, M
    Prasad, N
    Badve, S
    MODERN PATHOLOGY, 2006, 19 : 195A - 195A
  • [24] 5′-inositol phosphatase 2 (SHIP2) is over-expressed in endometrial cancer
    Sever, M
    Tandon, M
    Prasad, N
    Badve, S
    LABORATORY INVESTIGATION, 2006, 86 : 195A - 195A
  • [25] SHIP1 Regulates MSC Numbers and Their Osteolineage Commitment by Limiting Induction of the PI3K/Akt/β-Catenin/Id2 Axis
    Iyer, Sonia
    Viernes, Dennis R.
    Chisholm, John D.
    Margulies, Bryan S.
    Kerr, William G.
    STEM CELLS AND DEVELOPMENT, 2014, 23 (19) : 2336 - 2351
  • [26] Reversible Ser/Thr SHIP phosphorylation: A new paradigm in phosphoinositide signalling? Targeting of SHIP1/2 phosphatases may be controlled by phosphorylation on Ser and Thr residues
    Edimo, William's Elong
    Janssens, Veerle
    Waelkens, Etienne
    Erneux, Christophe
    BIOESSAYS, 2012, 34 (08) : 634 - 642
  • [27] The PI3K effector Arap3 interacts with the PI(3,4,5)P3 phosphatase SHIP2 in a SAM domain-dependent manner
    Raaijmakers, Judith H.
    Deneubourg, Laurence
    Rehmann, Holger
    de Koning, John
    Zhang, Zhongchun
    Krugmann, Sonja
    Erneux, Christophe
    Bos, Johannes L.
    CELLULAR SIGNALLING, 2007, 19 (06) : 1249 - 1257
  • [28] Human Cytomegalovirus Mediates Unique Monocyte-to-Macrophage Differentiation through the PI3K/SHIP1/Akt Signaling Network
    Cojohari, Olesea
    Mahmud, Jamil
    Altman, Aaron M.
    Peppenelli, Megan A.
    Miller, Michael J.
    Chan, Gary C.
    VIRUSES-BASEL, 2020, 12 (06):
  • [29] DAP12 signaling in osteoclasts and macrophages is regulated by SHIP1 and PI3K binding to the ITAM of DAP12
    Peng, Qisheng
    Humphrey, Mary Beth
    JOURNAL OF IMMUNOLOGY, 2009, 182
  • [30] Regulation of PtdIns(3,4,5)P3/Akt signalling by inositol polyphosphate 5-phosphatases
    Eramo, Matthew J.
    Mitchell, Christina A.
    BIOCHEMICAL SOCIETY TRANSACTIONS, 2016, 44 : 240 - 252
12345