Mechanisms of IgE Inflammation

被引:53
作者
Rosenwasser, Lanny J. [1 ]
机构
[1] Univ Missouri, Sch Med, Childrens Mercy Hosp, Div Allergy Immunol, Kansas City, MO 64108 USA
关键词
IgE; Fc receptors; Inflammation; FC-EPSILON-RI; HUMAN-IMMUNOGLOBULIN-E; HIGH-AFFINITY; DENDRITIC CELLS; BINDING-SITE; SERUM IGE; RECEPTOR; ANTIBODIES; CD23; EXPRESSION;
D O I
10.1007/s11882-011-0179-6
中图分类号
R392 [医学免疫学];
学科分类号
100102 ;
摘要
The prevalence of diseases such as allergic asthma and rhinitis continues to increase in the United States, affecting millions of people. It is well-established that allergy contributes to the pathogenesis of most asthma, especially in children and young adults. Despite current therapy (eg, inhaled corticosteroids, anti-leukotrienes, and bronchodilators), patients with moderate to severe asthma remain symptomatic and experience frequent exacerbations of disease requiring oral corticosteroids, emergency department treatments, and hospitalizations. Allergic diseases are traditionally referred to as immediate or type 1 hypersensitivity reactions, with IgE as a critical factor. IgE is involved in allergic inflammation, especially in early-phase response, but it may also be involved in the late-phase allergic response. A direct correlation between serum IgE levels and asthma exists. As logarithm IgE values increase, asthma prevalence increases linearly, even in patients who are categorized as having nonallergic asthma. In addition, there is a significant, although low association in allergic rhinitis with IgE levels and positive skin test reactivity to pollens. Recent advances in our understanding of the role of IgE in allergic inflammation have led to the development of a monoclonal antibody to IgE that reduces IgE levels, thereby reducing allergic inflammation. This review aims to provide an overview of the basic science of the IgE molecule and the clinical efficacy of anti-IgE therapy in allergic and asthmatic diseases.
引用
收藏
页码:178 / 183
页数:6
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