Translational Research Opportunities Regarding Homologous Recombination in Ovarian Cancer

被引:6
作者
Romeo, Margarita [1 ,2 ]
Carlos Pardo, Juan [1 ]
Martinez-Cardus, Anna [3 ]
Martinez-Balibrea, Eva [4 ]
Quiroga, Vanesa [1 ]
Martinez-Roman, Sergio [5 ]
Sole, Francesc [6 ]
Margeli, Mireia [1 ]
Mesia, Ricard [1 ]
机构
[1] Inst Catala Oncol Badalona, B ARGO Grp, Med Oncol Dept, Carretera Canyet S-N, Badalona 08916, Spain
[2] Univ Autonoma Barcelona, Campus UAB,Placa Civ, Bellaterra 08193, Spain
[3] Germans Trias & Pujol Fdn IGTP, Hlth Sci Res Inst, B ARGO Grp, Carretera Canyet S-N, Badalona 08916, Spain
[4] Hlth Sci Res Inst Germans Trias & Pujo IGTP, Program Predict & Personalized Canc Med PMPPC, Inst Catala Oncol Badalona, Program Canc Therapeut Resistance ProCURE, Carretera Can Ruti,Cami Escoles S-N, Badalona 08916, Spain
[5] Hosp Badalona Germans Trias & Pujol, Gynecol Dept, Badalona 08916, Spain
[6] Inst Recerca Leucemia Josep Carreras, Badalona 08916, Spain
关键词
ovarian cancer; high-grade serous ovarian cancer; deficient homologous recombination; PARP inhibitors; BRCA1; BRCA2; mechanisms of resistance; PLATINUM-SENSITIVE OVARIAN; OLAPARIB MAINTENANCE THERAPY; RANDOMIZED PHASE-2 TRIAL; PARP INHIBITOR RUCAPARIB; POLY(ADP-RIBOSE) POLYMERASE; DNA-REPAIR; BREAST-CANCER; SEROUS OVARIAN; FALLOPIAN-TUBE; DOUBLE-BLIND;
D O I
10.3390/ijms19103249
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Homologous recombination (HR) is a DNA repair pathway that is deficient in 50% of high-grade serous ovarian carcinomas (HGSOC). Deficient HR (DHR) constitutes a therapeutic opportunity for these patients, thanks to poly (ADP-ribose) polymerases (PARP) inhibitors (PARPi; olaparib, niraparib, and rucaparib are already commercialized). Although initially, PARPi were developed for patients with BRCA1/2 mutations, robust clinical data have shown their benefit in a broader population without DHR. This breakthrough in daily practice has raised several questions that necessitate further research: How can populations that will most benefit from PARPi be selected? At which stage of ovarian cancer should PARPi be used? Which strategies are reasonable to overcome PARPi resistance? In this paper, we present a summary of the literature and discuss the present clinical research involving PARPi (after reviewing ClinicalTrials.gov) from a translational perspective. Research into the functional biomarkers of DHR and clinical trials testing PARPi benefits as first-line setting or rechallenge are currently ongoing. Additionally, in the clinical setting, only secondary restoring mutations of BRCA1/2 have been identified as events inducing resistance to PARPi. The clinical frequency of this and other mechanisms that have been described in preclinics is unknown. It is of great importance to study mechanisms of resistance to PARPi to guide the clinical development of drug combinations.
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页数:18
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