Advances in immunology: Tolerance and autoimmunity.

[1] An autoimmune disease, APECED, caused by mutations in a novel gene featuring two PHD-type zinc-finger domains [J]. NATURE GENETICS, 1997, 17 (04) : 399 - 403

[2] Blockade of T lymphocyte costimulation with cytotoxic T lymphocyte-associated antigen 4-immunoglobulin (CTLA4Ig) reverses the cellular pathology of psoriatic plaques, including the activation of keratinocytes, dendritic cells, and endothelial cells [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 192 (05) : 681 - 693

[3] A range of CD4 T cell tolerance: Partial inactivation to organ-specific antigen allows nondestructive thyroiditis or insulitis [J]. IMMUNITY, 1997, 7 (02) : 255 - 271

[4] Self-reactive B cells are not eliminated or inactivated by autoantigen expressed on thyroid epithelial cells [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 186 (12) : 2005 - 2012

[5] Mechanisms of disease: Molecular mimicry and autoimmunity. [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1999, 341 (27) : 2068 - 2074

[6] High frequency of autoreactive myelin proteolipid protein-specific T cells in the periphery of naive mice: Mechanisms of selection of the self-reactive repertoire [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 191 (05) : 761 - 770

[7] [Anonymous], 1999, Neurology, V53, P457

[8] IL-10 is a key cytokine in psoriasis -: Proof of principle by IL-10 therapy:: A new therapeutic approach [J]. JOURNAL OF CLINICAL INVESTIGATION, 1998, 101 (04) : 783 - 794

[9] The NOD mouse model of type 1 diabetes: As good as it gets? [J]. NATURE MEDICINE, 1999, 5 (06) : 601 - 604

[10] Macrophage-dependent apoptosis of CD4(+) T lymphocytes from HIV-infected individuals is mediated by FasL and tumor necrosis factor [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 185 (01) : 55 - 64

[1] An autoimmune disease, APECED, caused by mutations in a novel gene featuring two PHD-type zinc-finger domains [J]. NATURE GENETICS, 1997, 17 (04) : 399 - 403

[2] Blockade of T lymphocyte costimulation with cytotoxic T lymphocyte-associated antigen 4-immunoglobulin (CTLA4Ig) reverses the cellular pathology of psoriatic plaques, including the activation of keratinocytes, dendritic cells, and endothelial cells [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 192 (05) : 681 - 693

[3] A range of CD4 T cell tolerance: Partial inactivation to organ-specific antigen allows nondestructive thyroiditis or insulitis [J]. IMMUNITY, 1997, 7 (02) : 255 - 271

[4] Self-reactive B cells are not eliminated or inactivated by autoantigen expressed on thyroid epithelial cells [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 186 (12) : 2005 - 2012

[5] Mechanisms of disease: Molecular mimicry and autoimmunity. [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1999, 341 (27) : 2068 - 2074

[6] High frequency of autoreactive myelin proteolipid protein-specific T cells in the periphery of naive mice: Mechanisms of selection of the self-reactive repertoire [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 191 (05) : 761 - 770

[7] [Anonymous], 1999, Neurology, V53, P457

[8] IL-10 is a key cytokine in psoriasis -: Proof of principle by IL-10 therapy:: A new therapeutic approach [J]. JOURNAL OF CLINICAL INVESTIGATION, 1998, 101 (04) : 783 - 794

[9] The NOD mouse model of type 1 diabetes: As good as it gets? [J]. NATURE MEDICINE, 1999, 5 (06) : 601 - 604

[10] Macrophage-dependent apoptosis of CD4(+) T lymphocytes from HIV-infected individuals is mediated by FasL and tumor necrosis factor [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1997, 185 (01) : 55 - 64