Long non-coding RNA H19 promotes the proliferation of fibroblasts in keloid scarring

被引:35
作者
Zhang, Jie [1 ]
Liu, Cai Yue [2 ]
Wan, Yun [3 ]
Peng, Li [3 ]
Li, Wen Fang [4 ]
Qiu, Jia Xuan [1 ]
机构
[1] Nanchang Univ, Dept Oral & Maxillofacial Surg, Affiliated Hosp 1, 17 Yongwai Main St, Nanchang 330006, Jiangxi, Peoples R China
[2] Second Mil Med Univ, Changzheng Hosp, Dept Plast Surg, Shanghai 200003, Peoples R China
[3] Nanchang Univ, Dept Plast Surg, Affiliated Hosp 1, Nanchang 330006, Jiangxi, Peoples R China
[4] Wenzhou Heping Plast Surg Hosp, Dept Plast Surg, 312 East Ring Rd, Wenzhou 325000, Zhejiang, Peoples R China
基金
中国国家自然科学基金;
关键词
keloid; long non-coding RNA H19; proliferation; mammalian target of rapamycin; vascular endothelial growth factor; NORMAL HUMAN KERATINOCYTES; GROWTH-FACTOR FAMILY; HYPERTROPHIC SCARS; BIOLOGY; CANCER; VEGF; EXPRESSION; MANAGEMENT; RECEPTORS; MTOR;
D O I
10.3892/ol.2016.4931
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
The expression of the long non-coding RNA (lncRNA) H19 is associated with proliferation in tumors. In order to investigate whether H19 may additionally mediate the proliferation of fibroblasts in human keloid disease, the present study collected samples from 24 subjects, including 8 with keloids, 8 with normal scars and 8 normal skin controls. Reverse transcription-polymerase chain reaction revealed that H19 levels were markedly increased in human keloids compared with normal scars and normal skin controls (P=0.017). In order to identify a potential role for H19 in the proliferative activity of human keloid fibroblasts, small interfering (si)RNA-mediated silencing experiments were performed. H19 siRNA treatment markedly inhibited the proliferation of keloid fibroblasts, as assessed by 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide assay (P=0.008). In order to identify the signaling mediators that are regulated by H19 in keloid fibroblasts, the expression levels of mammalian target of rapamycin (mTOR) and vascular endothelial growth factor (VEGF) were examined using western blotting. The results confirmed that knockdown of H19 inhibited mTOR and VEGF expression. In summary, the results indicate that H19 may be associated with increased proliferative activity of keloid fibroblasts, which may be mediated by mTOR and VEGF.
引用
收藏
页码:2835 / 2839
页数:5
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