Epigenetics and DOHaD: from basics to birth and beyond

被引:234
作者
Bianco-Miotto, T. [1 ,2 ]
Craig, J. M. [3 ]
Gasser, Y. P. [3 ]
van Dijk, S. J. [4 ]
Ozanne, S. E. [5 ]
机构
[1] Univ Adelaide, Sch Agr Food & Wine, Waite Res Inst, Adelaide, SA 5005, Australia
[2] Univ Adelaide, Robinson Res Inst, Adelaide, SA 5005, Australia
[3] Univ Melbourne, Murdoch Childrens Res Inst, Royal Childrens Hosp, Dept Paediat, Parkville, Vic, Australia
[4] CSIRO Hlth & Biosecur, N Ryde, NSW, Australia
[5] Univ Cambridge, Addenbrookes Hosp, MRC Metab Dis Unit, Metab Res Labs, Cambridge, England
基金
英国医学研究理事会;
关键词
animal models; diet; epigenetics; metabolic health; microbiome; EPIGENOME-WIDE ASSOCIATION; BODY-MASS INDEX; DNA METHYLATION; GUT MICROBIOTA; PROMOTER METHYLATION; WAIST CIRCUMFERENCE; GENE; METABOLITES; HEALTH; OBESITY;
D O I
10.1017/S2040174417000733
中图分类号
R1 [预防医学、卫生学];
学科分类号
1004 ; 120402 ;
摘要
Developmental origins of health and disease (DOHaD) is the study of how the early life environment can impact the risk of chronic diseases from childhood to adulthood and the mechanisms involved. Epigenetic modifications such as DNA methylation, histone modifications and non-coding RNAs are involved in mediating how early life environment impacts later health. This review is a summary of the Epigenetics and DOHaD workshop held at the 2016 DOHaD Society of Australia and New Zealand Conference. Our extensive knowledge of how the early life environment impacts later risk for chronic disease would not have been possible without animal models. In this review we highlight some animal model examples that demonstrate how an adverse early life exposure results in epigenetic and gene expression changes that may contribute to increased risk of chronic disease later in life. Type 2 diabetes and cardiovascular disease are chronic diseases with an increasing incidence due to the increased number of children and adults that are obese. Epigenetic changes such as DNA methylation have been shown to be associated with metabolic health measures and potentially predict future metabolic health status. Although more difficult to elucidate in humans, recent studies suggest that DNA methylation may be one of the epigenetic mechanisms that mediates the effects of early life exposures on later life risk of obesity and obesity related diseases. Finally, we discuss the role of the microbiome and how it is a new player in developmental programming and mediating early life exposures on later risk of chronic disease.
引用
收藏
页码:513 / 519
页数:7
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