Role of air pollutants mediated oxidative stress in respiratory diseases

被引：21

作者：

Traina, Giovanni ^{[1
]}

Bolzacchini, Ezio ^{[2
]}

Bonini, Maira ^{[3
]}

Contini, Daniele ^{[4
]}

Mantecca, Paride ^{[2
]}

Caimmi, Silvia Maria Elena ^{[5
]}

Licari, Amelia ^{[5
,6
]}

机构：

[1] ASST Melegnano Martesana, Melzo, Italy

[2] Univ Milano Bicocca, Dept Earth & Environm POLARIS, Milan, Italy

[3] Milan Hyg & Publ Hlth Serv, Agcy Hlth Protect Metropolitan Area, Milan West, Italy

[4] CNR, ISAC, Ist Sci Atmosfera & Clima, Lecce, Italy

[5] Fdn IRCCS Policlin San Matteo, Pediat Clin, Pavia, Italy

[6] Univ Pavia, Dept Clin Surg Diagnost & Pediat Sci, Pavia, Italy

来源：

PEDIATRIC ALLERGY AND IMMUNOLOGY | 2022年 / 33卷

关键词：

asthma; fractionated exhaled nitric oxide; indoor air quality; oxidative stress; particulate air pollution;

D O I：

10.1111/pai.13625

中图分类号：

R392 [医学免疫学];

学科分类号：

100102 ;

摘要：

Airborne particulate (PM) components from fossil fuel combustion can induce oxidative stress initiated by reactive oxygen species (ROS) that are strongly correlated with airway inflammation and asthma. A valid biomarker of airway inflammation is fractionated exhaled nitric oxide (FENO). The oxidative potential of PM2.5 can be evaluated with the dithiothreitol (DTT) dosage, which represents both ROS chemically produced and intracellular ROS of macrophages. This correlates with quality indicators of the internal environment and ventilation strategies such as dilution and removal of airborne contaminants.

引用

页码：38 / 40

页数：3

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