Role of dysregulated apoptosis in atopic dermatitis

被引：22

作者：

Trautmann, A ^{[1
]}

Akdis, M ^{[1
]}

Blaser, K ^{[1
]}

Akdis, CA ^{[1
]}

机构：

[1] Swiss Inst Allergy & Asthma Res, CH-7270 Davos, Switzerland

来源：

APOPTOSIS | 2000年 / 5卷 / 05期

关键词：

T cells; keratinocytes; eosinophils; eczematous dermatitis; eczema;

D O I：

10.1023/A:1009620329213

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Atopic dermatitis is a chronic inflammatory skin disease with a complex immune dysregulation and interplay of genetic, environmental and psychological factors. Activation and skin-selective homing of peripherial-blood T cells, and effector functions in the skin, represent sequential events in the pathogenesis. Dysregulated apoptosis in skin-homing T cells, eosinophils and keratino-cytes contributes to the elicitation and persistence of atopic derrmatitis.

引用

页码：425 / 429

页数：5

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