p53 null mice, nothing to lose?

被引：0

作者：

Gordon, AJE

机构：

来源：

TRENDS IN GENETICS | 1996年 / 12卷 / 11期

关键词：

D O I：

10.1016/0168-9525(96)20012-5

中图分类号：

Q3 [遗传学];

学科分类号：

071007 ; 090102 ;

摘要：

引用

页码：444 / 444

页数：1

共 8 条

[1] THYMOCYTE APOPTOSIS INDUCED BY P53-DEPENDENT AND INDEPENDENT PATHWAYS [J].

CLARKE, AR ;

PURDIE, CA ;

HARRISON, DJ ;

MORRIS, RG ;

BIRD, CC ;

HOOPER, ML ;

WYLLIE, AH .

NATURE, 1993, 362 (6423) :849-852

[2] MICE DEFICIENT FOR P53 ARE DEVELOPMENTALLY NORMAL BUT SUSCEPTIBLE TO SPONTANEOUS TUMORS [J].

DONEHOWER, LA ;

HARVEY, M ;

SLAGLE, BL ;

MCARTHUR, MJ ;

MONTGOMERY, CA ;

BUTEL, JS ;

BRADLEY, A .

NATURE, 1992, 356 (6366) :215-221

[3] THE DOMINATING EFFECT OF MUTANT P53 [J].

HANN, BC ;

LANE, DP .

NATURE GENETICS, 1995, 9 (03) :221-222

[4] A MUTANT P53 TRANSGENE ACCELERATES TUMOR-DEVELOPMENT IN HETEROZYGOUS BUT NOT NULLIZYGOUS P53 DEFICIENT MICE [J].

HARVEY, M ;

VOGEL, H ;

MORRIS, D ;

BRADLEY, A ;

BERNSTEIN, A ;

DONEHOWER, LA .

NATURE GENETICS, 1995, 9 (03) :305-311

[5] FUNCTIONAL INACTIVATION OF GENES BY DOMINANT NEGATIVE MUTATIONS [J].

HERSKOWITZ, I .

NATURE, 1987, 329 (6136) :219-222

[6]

KNUDSON AG, 1985, CANCER RES, V45, P1437

[7] P53 IS REQUIRED FOR RADIATION-INDUCED APOPTOSIS IN MOUSE THYMOCYTES [J].

LOWE, SW ;

SCHMITT, EM ;

SMITH, SW ;

OSBORNE, BA ;

JACKS, T .

NATURE, 1993, 362 (6423) :847-849

[8]

1994, NAT GENET, V8, P319

← 1 →