Resolving differences in GABAA receptor mutant mouse studies

被引：50

作者：

Crestani, F

Martini, JR

Möhler, H

Rudolph, U

机构：

[1] Univ Zurich, Inst Pharmacol & Toxicol, CH-8057 Zurich, Switzerland

[2] Swiss Fed Inst Technol, CH-8057 Zurich, Switzerland

[3] F Hoffmann LaRoche, Pharma Res, CH-4070 Basel, Switzerland

来源：

NATURE NEUROSCIENCE | 2000年 / 3卷 / 11期

关键词：

D O I：

10.1038/80553

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

引用

页码：1059 / 1059

页数：1

共 3 条

[1] Sedative but not anxiolytic properties of benzodiazepines ave mediated by the GABAA receptor α1 subtype [J].

McKernan, RM ;

Rosahl, TW ;

Reynolds, DS ;

Sur, C ;

Wafford, KA ;

Atack, JR ;

Farrar, S ;

Myers, J ;

Cook, G ;

Ferris, P ;

Garrett, L ;

Bristow, L ;

Marshall, G ;

Macaulay, A ;

Brown, N ;

Howell, O ;

Moore, KW ;

Carling, RW ;

Street, LJ ;

Castro, JL ;

Ragan, CI ;

Dawson, GR ;

Whiting, PJ .

NATURE NEUROSCIENCE, 2000, 3 (06) :587-592

[2] Benzodiazepine actions mediated by specific γ-aminobutyric acidA receptor subtypes [J].

Rudolph, U ;

Crestani, F ;

Benke, D ;

Brünig, I ;

Benson, JA ;

Fritschy, JM ;

Martin, JR ;

Bluethmann, H ;

Möhler, H .

NATURE, 1999, 401 (6755) :796-800

[3] Designer genes and anti-anxiety drugs [J].

Tecott, LH .

NATURE NEUROSCIENCE, 2000, 3 (06) :529-530

← 1 →