Non-homologous end joining as a mechanism of DNA repair

被引：47
作者：
Barnes, DE [1 ]
机构：
[1] Imperial Canc Res Fund, Clare Hall Labs, S Mimms EN6 3LD, Herts, England
来源：
CURRENT BIOLOGY | 2001年 / 11卷 / 12期
关键词：
D O I：
10.1016/S0960-9822(01)00279-2
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
引用
收藏
页码：R455 / R457
页数：3
相关论文
共 6 条
[1]   Partners and pathways - repairing a double-strand break [J].
Haber, JE .
TRENDS IN GENETICS, 2000, 16 (06) :259-264
[2]   Binding of inositol phosphate to DNA-PK and stimulation of double-strand break repair [J].
Hanakahi, LA ;
Bartlet-Jones, M ;
Chappell, C ;
Pappin, D ;
West, SC .
CELL, 2000, 102 (06) :721-729
[3]   DNA double strand break repair in mammalian cells [J].
Karran, P .
CURRENT OPINION IN GENETICS & DEVELOPMENT, 2000, 10 (02) :144-150
[4]   DNA double-strand breaks: signaling, repair and the cancer connection [J].
Khanna, KK ;
Jackson, SP .
NATURE GENETICS, 2001, 27 (03) :247-254
[5]   Sterility of Drosophila with mutations in the bloom syndrome gene -: Complementation by Ku70 [J].
Kusano, K ;
Johnson-Schlitz, DM ;
Engels, WR .
SCIENCE, 2001, 291 (5513) :2600-2602
[6]   Artemis, a novel DNA double-strand break repair/V(D)J recombination protein, is mutated in human severe combined immune deficiency [J].
Moshous, D ;
Callebaut, I ;
de Chasseval, R ;
Corneo, B ;
Cavazzana-Calvo, M ;
Le Deist, F ;
Tezcan, I ;
Sanal, O ;
Bertrand, Y ;
Philippe, N ;
Fischer, A ;
de Villartay, JP .
CELL, 2001, 105 (02) :177-186
1