Systems biological approaches to the cardiac signaling network

被引:9
作者
Kang, Jun Hyuk [2 ]
Lee, Ho-Sung [2 ]
Kang, Yun-Won [1 ]
Cho, Kwang-Hyun [1 ]
机构
[1] Korea Adv Inst Sci & Technol, Dept Bio & Brain Engn, 291 Daehak Ro, Daejeon 305338, South Korea
[2] Korea Adv Inst Sci & Technol, Grad Sch Med Sci & Engn, Daejeon 305338, South Korea
基金
新加坡国家研究基金会;
关键词
cardiac signaling network; heart failure; mathematical modeling; molecular dynamics analysis; personalized therapy; systems biology; PHOSPHOINOSITIDE 3-KINASE PATHWAY; ISCHEMIA-REPERFUSION INJURY; DIASTOLIC HEART-FAILURE; N-TERMINAL KINASES; TRANSGENIC MICE; IN-VIVO; CALCINEURIN ACTIVITY; BETA(2)-ADRENERGIC RECEPTOR; BETA(1)-ADRENERGIC RECEPTOR; PERSONALIZED MEDICINE;
D O I
10.1093/bib/bbv039
中图分类号
Q5 [生物化学];
学科分类号
071010 ; 081704 ;
摘要
Recent systems biological studies of cardiac systems have greatly advanced our understanding of cardiac physiology with a particular focus on the excitation-contraction coupling. With these advancements, there is a growing interest in systems analysis of the cardiac signaling network because its dynamical property is closely associated with cardiac diseases. In this article, we review recent attempts at computational modeling of the cardiac signaling network and provide a system-level perspective on the analysis of the large-scale cardiac signaling network. We discuss why the systems biological approach is useful and what novel insights it can provide for the development of personalized therapeutic strategies for cardiac diseases in the post-genomic era.
引用
收藏
页码:419 / 428
页数:10
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