Harnessing the predictive power of preclinical models for oncology drug development

被引:50
作者
Honkala, Alexander [1 ]
Malhotra, Sanjay, V [1 ,2 ]
Kummar, Shivaani [2 ,3 ]
Junttila, Melissa R. [4 ]
机构
[1] Oregon Hlth & Sci Univ, Dept Cell Dev & Canc Biol, Portland, OR 97201 USA
[2] Oregon Hlth & Sci Univ, Ctr Expt Therapeut, Knight Canc Inst, Portland, OR 97201 USA
[3] Oregon Hlth & Sci Univ, Div Hematol & Med Oncol, Portland, OR 97201 USA
[4] ORIC Pharmaceut, San Francisco, CA 94080 USA
关键词
CELL LUNG-CANCER; ENGINEERED MOUSE MODELS; PATIENT-DERIVED XENOGRAFTS; ACQUIRED-RESISTANCE; INHIBITOR RESISTANCE; TUMOR HETEROGENEITY; PRECISION MEDICINE; SIGNALING PATHWAY; ONCOGENIC BRAF; RAS MUTATIONS;
D O I
10.1038/s41573-021-00301-6
中图分类号
Q81 [生物工程学(生物技术)]; Q93 [微生物学];
学科分类号
071005 ; 0836 ; 090102 ; 100705 ;
摘要
Recent progress in understanding the molecular basis of cellular processes, identification of promising therapeutic targets and evolution of the regulatory landscape makes this an exciting and unprecedented time to be in the field of oncology drug development. However, high costs, long development timelines and steep rates of attrition continue to afflict the drug development process. Lack of predictive preclinical models is considered one of the key reasons for the high rate of attrition in oncology. Generating meaningful and predictive results preclinically requires a firm grasp of the relevant biological questions and alignment of the model systems that mirror the patient context. In doing so, the ability to conduct both forward translation, the process of implementing basic research discoveries into practice, as well as reverse translation, the process of elucidating the mechanistic basis of clinical observations, greatly enhances our ability to develop effective anticancer treatments. In this Review, we outline issues in preclinical-to-clinical translatability of molecularly targeted cancer therapies, present concepts and examples of successful reverse translation, and highlight the need to better align tumour biology in patients with preclinical model systems including tracking of strengths and weaknesses of preclinical models throughout programme development.
引用
收藏
页码:99 / 114
页数:16
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