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SARS-CoV-2'nin Olasi Noroinvazyon Mekanizmalari
被引:1
|作者:
Sahin, Ali
[1
]
Korkunc, Seval Kubra
[2
]
Kurtoglu, Fatima Hacer
[3
]
Taskiran, Hilal
[3
]
Ekmekci, Betigul
[4
]
Babayev, Huseyn
[1
]
Ekmekci, Ahmet Hakan
[5
]
机构:
[1] Selcuk Univ, Fac Med, Konya, Turkey
[2] Koc Univ, Inst Hlth Sci, Dept Immunol, Istanbul, Turkey
[3] Koc Univ, Inst Hlth Sci, Dept Cellular & Mol Med, Istanbul, Turkey
[4] Bahcesehir Univ, Fac Med, Istanbul, Turkey
[5] Selcuk Univ, Fac Med, Dept Neurol, Konya, Turkey
来源:
TURKISH JOURNAL OF IMMUNOLOGY
|
2022年
/
10卷
/
02期
关键词:
SARS-CoV-2;
antiviral immunity;
neurology;
neuroinvasion;
COVID-19;
ACUTE RESPIRATORY SYNDROME;
T-CELL RESPONSES;
SARS CORONAVIRUS;
NEUROLOGICAL MANIFESTATIONS;
LYMPHATIC DRAINAGE;
IMMUNE-RESPONSE;
CEREBRAL EDEMA;
COVID-19;
INFECTION;
SYSTEM;
D O I:
10.4274/tji.galenos.2022.80774
中图分类号:
R392 [医学免疫学];
Q939.91 [免疫学];
学科分类号:
100102 ;
摘要:
The severe acute respiratory syndrome coronavirus-2, a coronavirus, is known to cause acute respiratory distress syndrome and a range of nonrespiratory effects, particularly in elderly male patients with underlying health conditions such overweight, diabetes, and hypertension. The coronavirus disease-2019 sequelae include multiple organ failure and neurological issues, and these prior health issues are linked to endothelial dysfunction. Although inhalation is the most frequent mode of infection, this virus has also been discovered in neurons, cerebrospinal fluid, the choroid plexus, and meningeal vasculature.
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页码:69 / 76
页数:8
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