Microvascular disease and its role in the brain and cardiovascular system: a potential role for uric acid as a cardiorenal toxin

被引:65
作者
Kanbay, Mehmet [1 ]
Sanchez-Lozada, Laura-Gabriela [2 ]
Franco, Martha [2 ]
Madero, Magdalena [2 ]
Solak, Yalcin [3 ]
Rodriguez-Iturbe, Bernardo [4 ,5 ]
Covic, Adrian [6 ]
Johnson, Richard J. [7 ]
机构
[1] Gulhane Mil Med Acad, Dept Med, Div Nephrol, Ankara, Turkey
[2] INC Ignacio Chavez, Dept Nephrol, Mexico City, DF, Mexico
[3] Selcuk Univ, Meram Sch Med, Dept Med, Div Nephrol, Konya, Turkey
[4] Univ Zulia, IVIC Zulia, Maracaibo 4011, Venezuela
[5] Univ Hosp, Serv Nefrol, Maracaibo, Venezuela
[6] Gr T Popa Univ Med & Pharm, CI PARHON Univ Hosp, Dialysis & Renal Transplant Ctr, Nephrol Clin, Iasi, Romania
[7] Univ Colorado, Div Renal Dis & Hypertens, Denver, CO 80202 USA
关键词
arteriolosclerosis; autoregulation; chronic kidney disease; fructose; uric acid; SALT-SENSITIVE HYPERTENSION; MUSCLE-CELL-PROLIFERATION; CHRONIC KIDNEY-DISEASE; OXIDATIVE STRESS; BLOOD-PRESSURE; COGNITIVE FUNCTION; RENAL INJURY; GLOMERULAR HYPERTENSION; METABOLIC SYNDROME; FRUCTOSE INTAKE;
D O I
10.1093/ndt/gfq635
中图分类号
R3 [基础医学]; R4 [临床医学];
学科分类号
1001 ; 1002 ; 100602 ;
摘要
Arteriolosclerosis (microvascular disease) may have a key role not only in driving salt-sensitive hypertension but also in mediating the development of chronic kidney disease, vascular dementia, stroke and coronary heart disease. In this paper, we review the evidence that these latter conditions result from the altered autoregulation that occurs when arterioles become diseased. We also discuss the increasing evidence that dietary intake of sugars rich in fructose may be driving the development of microvascular disease as a consequence of raising intracellular uric acid. We hypothesize that the treatment of microvascular disease may require a multifaceted approach by utilizing agents which aim at blocking of the renin-angiotensin system, reducing oxidative stress, stimulating endothelial nitric oxide production and lowering uric acid levels. Paradoxically, agents that only stimulate nitric oxide, such as oestrogens, may increase the risk of poor outcomes if microvascular disease is not reversed.
引用
收藏
页码:430 / 437
页数:9
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