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- [43] Insulin-induced egr-1 and c-fos expression in 32D cells requires insulin receptor, Shc, and mitogen-activated protein kinase, but not insulin receptor substrate-1 and phosphatidylinositol 3-kinase activation (vol 271, pg 30222, 1996) JOURNAL OF BIOLOGICAL CHEMISTRY, 1997, 272 (05) : 3124 - 3124
- [46] Differential involvement of calmodulin-dependent protein kinase II-activated AP-1 and c-Jun N-terminal kinase-activated EGR-1 signaling pathways in tumor necrosis factor-α and lipopolysaccharide-induced CD44 expression in human monocytic cells JOURNAL OF BIOLOGICAL CHEMISTRY, 2005, 280 (29) : 26825 - 26837
- [49] Intracellular interleukin-1 receptor antagonist inhibits interleukin-i alpha induced inflammatory gene expression in CACO-2 cells through inhibition of p38 mitogen-activated protein kinase and NF-kappa B dependent pathways. ARTHRITIS AND RHEUMATISM, 2002, 46 (09): : S349 - S349
- [50] RETRACTION: Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells (Retraction of vol 73, pg 1549, 2008) MOLECULAR PHARMACOLOGY, 2016, 90 (01) : 63 - 63