Photoreceptor cells and RPE contribute to the development of diabetic retinopathy

被引:141
作者
Tonade, Deoye [1 ,5 ]
Kern, Timothy S. [1 ,2 ,3 ,4 ]
机构
[1] Case Western Reserve Univ, Dept Pharmacol, Cleveland, OH 44106 USA
[2] Vet Adm Med Ctr, Res Serv, 10701 East Blvd, Cleveland, OH 44106 USA
[3] Univ Calif Irvine, Gavin Herbert Eye Inst, Irvine, CA 92697 USA
[4] Vet Adm Med Ctr, Res Serv, Long Beach, CA 90822 USA
[5] Medpace Inc, Cincinnati, OH USA
关键词
Diabetes; Diabetic retinopathy; Outer retina; Photoreceptors; RPE; Vasculature; Phototransduction; Visual cycle; RETINAL-PIGMENT EPITHELIUM; ENDOTHELIAL GROWTH-FACTOR; OPTICAL COHERENCE TOMOGRAPHY; ALDOSE REDUCTASE INHIBITOR; INSULIN-RECEPTOR SUBSTRATE-2; S-CONE ELECTRORETINOGRAM; FREE-RADICAL PRODUCTION; TRANSGENIC MOUSE MODEL; OXIDATIVE STRESS; MACULAR EDEMA;
D O I
10.1016/j.preteyeres.2020.100919
中图分类号
R77 [眼科学];
学科分类号
100212 ;
摘要
Diabetic retinopathy (DR) is a leading cause of blindness. It has long been regarded as vascular disease, but work in the past years has shown abnormalities also in the neural retina. Unfortunately, research on the vascular and neural abnormalities have remained largely separate, instead of being integrated into a comprehensive view of DR that includes both the neural and vascular components. Recent evidence suggests that the most predominant neural cell in the retina (photoreceptors) and the adjacent retinal pigment epithelium (RPE) play an important role in the development of vascular lesions characteristic of DR. This review summarizes evidence that the outer retina is altered in diabetes, and that photoreceptors and RPE contribute to retinal vascular alterations in the early stages of the retinopathy. The possible molecular mechanisms by which cells of the outer retina might contribute to retinal vascular damage in diabetes also are discussed. Diabetes-induced alterations in the outer retina represent a novel therapeutic target to inhibit DR.
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页数:33
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