Targeting of BCR-ABL gene expression in K562 cells using cell-penetrating peptide nanocomplexes carrying siRNAs

被引：0
|
作者：
Vysochinskaya, V. [1 ]
Knyazev, N. A. [2 ]
Emelyanov, A. [2 ]
Terterov, I. [2 ]
Klimenko, V. [2 ]
Bogdanov, A. A. [2 ]
Dubina, M. [3 ]
机构：
[1] Minist Hlth Russian Federat, Smorodintsev Res Inst Influenza, Dept Mol Biol Viruses, St Petersburg, Russia
[2] St Petersburg Clin Res & Pract Ctr Specialized Ty, Sci Dept, St Petersburg, Russia
[3] State Res Inst Highly Pure Biopreparat, St Petersburg, Russia
来源：
ANNALS OF ONCOLOGY | 2021年 / 32卷
关键词：
D O I：
10.1016/j.annonc.2021.08.308
中图分类号：
R73 [肿瘤学];
学科分类号：
100214 ;
摘要：
30P
引用
收藏
页码：S370 / S370
页数：1
相关论文
共 50 条
  • [41] Cotreatment with apicidin overcomes TRAIL resistance via inhibition of Bcr-Abl signaling pathway in K562 leukemia cells
    Park, Soo-Jung
    Kim, Mi-Ju
    Kim, Hak-Bong
    Sohn, Hee-Young
    Bae, Joe-Ho
    Kang, Chi-Dug
    Kim, Sun-Hee
    EXPERIMENTAL CELL RESEARCH, 2009, 315 (11) : 1809 - 1818
  • [42] p53 mediates breakdown of the anti-apoptotic protein Bcr-Abl in K562 leukaemia cells
    DiBacco, A
    Cotter, TG
    BRITISH JOURNAL OF CANCER, 1998, 78 : 23 - 23
  • [43] Synergistic cytotoxicity effect of the combination of chitosan nanoencapsulated imatinib mesylate and quercetin in BCR-ABL positive K562 cells
    Kamyabi, Rohollah
    Jahandideh, Alireza
    Panahi, Negar
    Muhammadnejad, Samad
    IRANIAN JOURNAL OF BASIC MEDICAL SCIENCES, 2023, 26 (03) : 359 - 366
  • [44] BCR-ABL tyrosine kinase inhibition induces metabolic vulnerability by preventing the integrated stress response in K562 cells
    Kato, Yu
    Kunimasa, Kazuhiro
    Sugimoto, Yoshikazu
    Tomida, Akihiro
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2018, 504 (04) : 721 - 726
  • [45] ND-09 inhibits chronic myeloid leukemia K562 cell growth by regulating BCR-ABL signaling
    Liu, Yan-Hong
    Zhu, Man
    Lei, Pan-Pan
    Pan, Xiao-Yan
    Ma, Wei-Na
    ONCOLOGY REPORTS, 2021, 46 (01)
  • [46] Proteasome inhibition leads to significant reduction of Bcr-Abl expression and subsequent induction of apoptosis in K562 human chronic myelogenous leukemia cells
    Dou, QP
    McQuire, TF
    Peng, YB
    An, B
    JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS, 1999, 289 (02): : 781 - 790
  • [47] Adenovirus-mediated exogenous PTEN gene expression results in the loss of tumorigenicity in k562 cells by the inhibition of c-myb and bcr-abl function.
    Hwang, PH
    Yi, HK
    Park, MY
    Lee, DY
    BLOOD, 2002, 100 (11) : 203B - 203B
  • [48] P210 BCR-ABL IS COMPLEXED TO P160 BCR AND PH-P53 PROTEINS IN K562 CELLS
    CAMPBELL, ML
    LI, WJ
    ARLINGHAUS, RB
    ONCOGENE, 1990, 5 (05) : 773 - 776
  • [49] A novel pyridopyrimidine inhibitor of Abl kinase is a picomolar inhibitor of Bcr-Abl-driven K562 cells and is effective against STI571-resistant Bcr-Abl mutants
    Huron, DR
    Gorre, ME
    Kraker, AJ
    Sawyers, CL
    Rosen, N
    Moasser, MM
    CLINICAL CANCER RESEARCH, 2003, 9 (04) : 1267 - 1273
  • [50] BCR-ABL1 fusion gene acquisition of endothelial cells from K562 spent serum
    Kocana, C. C.
    Toprak, S. F.
    Sozer, S.
    EUROPEAN JOURNAL OF HUMAN GENETICS, 2020, 28 (SUPPL 1) : 500 - 500
12345