Role of the C-terminus in interaction of KCNQ2 and KCNQ3 K+ channels

被引:0
|
作者
Maljevic, S
Wuttke, T
Lerche, C
Seebohm, G
Alekov, AK
Busch, AE
Lerche, H
机构
[1] Univ Ulm, D-89081 Ulm, Germany
[2] Aventis Pharma Deutschland GmbH, D-65926 Frankfurt, Germany
关键词
D O I
暂无
中图分类号
Q6 [生物物理学];
学科分类号
071011 ;
摘要
引用
收藏
页码:548A / 548A
页数:1
相关论文
共 50 条
  • [1] C-terminal interaction of KCNQ2 and KCNQ3 K+ channels
    Maljevic, S
    Lerche, C
    Seebohm, G
    Alekov, AK
    Busch, AE
    Lerche, H
    JOURNAL OF PHYSIOLOGY-LONDON, 2003, 548 (02): : 353 - 360
  • [2] Expression and Localization of K+ Channels KCNQ2 and KCNQ3 in the Mammalian Cochlea
    Jin, Zhe
    Liang, Gui-Hua
    Cooper, Edward C.
    Jarlebark, Leif
    AUDIOLOGY AND NEURO-OTOLOGY, 2009, 14 (02) : 98 - 105
  • [3] KCNQ2/KCNQ3 K+ channels and the molecular pathogenesis of epilepsy:: implications for therapy
    Rogawski, MA
    TRENDS IN NEUROSCIENCES, 2000, 23 (09) : 393 - 398
  • [4] Reconstitution of muscarinic modulation of the KCNQ2/KCNQ3 K+ channels that underlie the neuronal M current
    Shapiro, MS
    Roche, JP
    Kaftan, EJ
    Cruzblanca, H
    Mackie, K
    Hille, B
    JOURNAL OF NEUROSCIENCE, 2000, 20 (05): : 1710 - 1721
  • [5] Effects of tetraethylammonium on KCNQ1-4 and on heteromeric KCNQ2/KCNQ3 and KCNQ2/KCNQ3(T323Y) potassium channels
    Hadley, JK
    Noda, M
    Selyanko, AA
    Wood, IC
    Abogadie, FC
    Jentsch, TJ
    Brown, DA
    BRITISH JOURNAL OF PHARMACOLOGY, 2000, 129
  • [6] KCNQ2 and KCNQ5 form heteromeric channels independent of KCNQ3
    Soh, Heun
    Springer, Kristen
    Doci, Klarita
    Balsbaugh, Jeremy L.
    Tzingounis, Anastasios V.
    PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2022, 119 (13)
  • [7] Moderate loss of function of cAMP-modulated KCNQ2 KCNQ3 K+ channels is sufficient to cause epilepsy
    Stein, V
    Schroeder, BC
    Kubisch, C
    Jentsch, TJ
    JOURNAL OF NEUROCHEMISTRY, 1999, 73 : S128 - S128
  • [8] Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy
    Björn C. Schroeder
    Christian Kubisch
    Valentin Stein
    Thomas J. Jentsch
    Nature, 1998, 396 : 687 - 690
  • [9] Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy
    Schroeder, BC
    Kubisch, C
    Stein, V
    Jentsch, TJ
    NATURE, 1998, 396 (6712) : 687 - 690
  • [10] Regulation of the voltage-gated K+ channels KCNQ2/3 and KCNQ3/5 by ubiquitination -: Novel role for Nedd4-2
    Ekberg, Jenny
    Schuetz, Friderike
    Boase, Natasha A.
    Conroy, Sarah-Jane
    Manning, Jantina
    Kumar, Sharad
    Poronnik, Philip
    Adams, David J.
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2007, 282 (16) : 12135 - 12142