Neurofascin as target of autoantibodies in Guillain-Barre syndrome

被引：41

作者：

Pruess, Harald ^{[1
]}

Schwab, Jan M. ^{[1
]}

Derst, Christian ^{[2
]}

Goertzen, Angelika ^{[3
]}

Veh, Ruediger W. ^{[2
]}

机构：

[1] Charite, Dept Neurol & Expt Neurol, D-10117 Berlin, Germany

[2] Charite, Inst Integrat Neuroanat, D-10115 Berlin, Germany

[3] St Josef Hosp, Dept Neurol, D-46045 Oberhausen, Germany

来源：

BRAIN | 2011年 / 134卷

关键词：

RANVIER; NODES;

D O I：

10.1093/brain/awq372

中图分类号：

R74 [神经病学与精神病学];

学科分类号：

摘要：

引用

页数：2

共 4 条

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[2] Disruption of neurofascin and gliomedin at nodes of Ranvier precedes demyelination in experimental allergic neuritis [J].

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BRAIN, 2009, 132 :260-273

[3] Neurofascin as a novel target for autoantibody-mediated axonal injury [J].

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Storch, Maria K. ;

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Hales, Kimberly ;

Woolley, David R. ;

Al-Hayani, Abdulmonem ;

Davies, Stephen N. ;

Rasband, Matthew N. ;

Olsson, Tomas ;

Moldenhauer, Anja ;

Velhin, Sviataslau ;

Hohlfeld, Reinhard ;

Meinl, Edgar ;

Linington, Christopher .

JOURNAL OF EXPERIMENTAL MEDICINE, 2007, 204 (10) :2363-2372

[4] Anti-GD1a antibodies activate complement and calpain to injure distal motor nodes of Ranvier in mice [J].

McGonigal, Rhona ;

Rowan, Edward G. ;

Greenshields, Kay N. ;

Halstead, Susan K. ;

Humphreys, Peter D. ;

Rother, Russell P. ;

Furukawa, Koichi ;

Willison, Hugh J. .

BRAIN, 2010, 133 :1944-1960

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