Notch is activated in RANKL-induced osteoclast differentiation and resorption

被引:13
作者
Duan, Li [1 ,2 ]
de Vos, Paul [3 ]
Fan, Mingwen [4 ]
Ren, Yijin [1 ]
机构
[1] Univ Groningen, Univ Med Ctr Groningen, Dept Orthodont, NL-9700 RB Groningen, Netherlands
[2] Univ Groningen, Univ Med Ctr Groningen, Inst Drug Explorat, NL-9700 RB Groningen, Netherlands
[3] Univ Groningen, Univ Med Ctr Groningen, Dept Pathol & Lab Sci, NL-9700 RB Groningen, Netherlands
[4] Wuhan Univ, Key Lab Oral Biomed Engn, Minist Educ, Sch & Hosp Stomatol, Wuhan, Peoples R China
来源
FRONTIERS IN BIOSCIENCE-LANDMARK | 2008年 / 13卷
关键词
RANKL; osteoclast; notch; NF-kappa B; Hes1; bone resorption;
D O I
10.2741/3210
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
The process of osteoclast differentiation and resorption is fine-tuned by signal pathways, which need to be further elucidated. The aim of this study was to explore the possible connections between NF-kappa B and Notch in RANKL-induced osteoclast activity. To this end, RANKL was used to stimulate mouse osteoclast precursor cell line RAW264.7. The number of multinucleated TRAP(+) osteoclasts was counted and the resorption area was measured. NF-kappa B transcriptional factor activity was determined by EMSA. Quantitative RT-PCR and Western blotting analysis were used to determine Hes1 (one of Notch signaling primary targets) mRNA and protein expressions respectively. Mature osteoclasts and bone resorption areas were detected in the present study. NF-kappa B activity was increased in RANKL-induced osteoclast differentiation and resorption. mRNA and protein expressions of Hes1 in RAW264.7 cells were up-regulated after RANKL stimulation. In conclusion, NF-kappa B signaling mediated RANKL-induced osteoclast differentiation and resorption, during which, Notch signaling was activated. Therefore, Notch could be a novel therapeutic target for bone resorption related diseases.
引用
收藏
页码:7064 / 7071
页数:8
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