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Role of the Purinergic P2Y2 Receptor in Pulmonary Hypertension
被引:6
作者:

Shihan, Mazen
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Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany

Novoyatleva, Tatyana
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Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany

Lehmeyer, Thilo
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Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany

Sydykov, Akylbek
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Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany

Schermuly, Ralph T.
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机构:
Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany
机构:
[1] Justus Liebig Univ Giessen, Excellence Cluster Cardiopulm Inst CPI, Univ Giessen & Marburg Lung Ctr UGMLC, German Ctr Lung Res DZL, Aulweg 130, D-35392 Giessen, Germany
关键词:
pulmonary arterial hypertension;
purinergic P2Y2 receptor;
pharmacological approach;
P2Y(2) NUCLEOTIDE RECEPTOR;
ENDOTHELIUM-DEPENDENT RELAXATION;
DECREASES BLOOD-PRESSURE;
VALVE INTERSTITIAL-CELLS;
SHEAR-STRESS;
ADENOSINE-TRIPHOSPHATE;
NITRIC-OXIDE;
INTRACELLULAR CA2+;
CYSTIC-FIBROSIS;
RENAL NA+;
D O I:
10.3390/ijerph182111009
中图分类号:
X [环境科学、安全科学];
学科分类号:
08 ;
0830 ;
摘要:
Pulmonary arterial hypertension (PAH), group 1 pulmonary hypertension (PH), is a fatal disease that is characterized by vasoconstriction, increased pressure in the pulmonary arteries, and right heart failure. PAH can be described by abnormal vascular remodeling, hyperproliferation in the vasculature, endothelial cell dysfunction, and vascular tone dysregulation. The disease pathomechanisms, however, are as yet not fully understood at the molecular level. Purinergic receptors P2Y within the G-protein-coupled receptor family play a major role in fluid shear stress transduction, proliferation, migration, and vascular tone regulation in systemic circulation, but less is known about their contribution in PAH. Hence, studies that focus on purinergic signaling are of great importance for the identification of new therapeutic targets in PAH. Interestingly, the role of P2Y2 receptors has not yet been sufficiently studied in PAH, whereas the relevance of other P2Ys as drug targets for PAH was shown using specific agonists or antagonists. In this review, we will shed light on P2Y receptors and focus more on the P2Y2 receptor as a potential novel player in PAH and as a new therapeutic target for disease management.
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