Activator protein 1 (Fos/Jun) functions in inflammatory bone and skin disease

被引:290
作者
Zenz, Rainer [1 ,2 ]
Eferl, Robert [1 ,2 ]
Scheinecker, Clemens [3 ]
Redlich, Kurt [3 ]
Smolen, Josef [3 ]
Schonthaler, Helia B. [4 ]
Kenner, Lukas [1 ,2 ,5 ]
Tschachler, Erwin [6 ]
Wagner, Erwin F. [4 ]
机构
[1] Ludwig Boltzmann Inst Canc Res, A-1090 Vienna, Austria
[2] Med Univ Vienna, Ctr Biomol Med & Pharm, A-1090 Vienna, Austria
[3] Med Univ Vienna, Div Rheumatol, A-1090 Vienna, Austria
[4] Res Inst Mol Pathol, A-1030 Vienna, Austria
[5] Med Univ Vienna, Clin Inst Pathol, A-1090 Vienna, Austria
[6] Med Univ Vienna, Dept Dermatol, A-1090 Vienna, Austria
基金
奥地利科学基金会;
关键词
D O I
10.1186/ar2338
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
Activator protein 1 (AP-1) (Fos/Jun) is a transcriptional regulator composed of members of the Fos and Jun families of DNA binding proteins. The functions of AP-1 were initially studied in mouse development as well as in the whole organism through conventional transgenic approaches, but also by gene targeting using knockout strategies. The importance of AP-1 proteins in disease pathways including the inflammatory response became fully apparent through conditional mutagenesis in mice, in particular when employing gene inactivation in a tissue-specific and inducible fashion. Besides the well-documented roles of Fos and Jun proteins in oncogenesis, where these genes can function both as tumor promoters or tumor suppressors, AP-1 proteins are being recognized as regulators of bone and immune cells, a research area termed osteoimmunology. In the present article, we review recent data regarding the functions of AP-1 as a regulator of cytokine expression and an important modulator in inflammatory diseases such as rheumatoid arthritis, psoriasis and psoriatic arthritis. These new data provide a better molecular understanding of disease pathways and should pave the road for the discovery of new targets for therapeutic applications.
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页数:10
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