Heat shock proteins and cardiovascular pathophysiology

被引:306
作者
Snoeckx, LHEH [1 ]
Cornelussen, RN [1 ]
Van Nieuwenhoven, FA [1 ]
Reneman, RS [1 ]
Van Der Vusse, GJ [1 ]
机构
[1] Univ Maastricht, Cardiovasc Res Inst, Dept Physiol, Fac Med, NL-6200 MD Maastricht, Netherlands
关键词
D O I
10.1152/physrev.2001.81.4.1461
中图分类号
Q4 [生理学];
学科分类号
071003 ;
摘要
In the eukaryotic cell an intrinsic mechanism is present providing the ability to defend itself against external stressors from various sources. This defense mechanism probably evolved from the presence of a group of chaperones, playing a crucial role in governing proper protein assembly, folding, and transport. Upregulation of the synthesis of a number of these proteins upon environmental stress establishes a unique defense system to maintain cellular protein homeostasis and to ensure survival of the cell. In the cardiovascular system this enhanced protein synthesis leads to a transient but powerful increase in tolerance to such endangering situations as ischemia, hypoxia, oxidative injury, and endotoxemia. These so-called heat shock proteins interfere with several physiological processes within several cell organelles and, for proper functioning, are translocated to different compartments following stress-induced synthesis. In this review we describe the physiological role of heat shock proteins and discuss their protective potential against various stress agents in the cardiovascular system.
引用
收藏
页码:1461 / 1497
页数:37
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