Drug induced angioedema (ACE-inhibitors and other)

被引：3

作者：

Fain, Olivier ^{[1
]}

Mekinian, Arsene ^{[1
]}

Gobert, Delphine ^{[1
]}

Khau, Cam-Anh ^{[1
]}

Javaud, Nicolas ^{[2
]}

机构：

[1] Univ Paris 06, Hop St Antoine, AP HP,Serv Med Interne, DHU i2B,Ctr References Angioedemes Kinines CREAK, F-75012 Paris, France

[2] Univ Paris 07, Hop Louis Mounier, AP HP, F-92700 Colombes, France

来源：

PRESSE MEDICALE | 2015年 / 44卷 / 01期

关键词：

ANGIOTENSIN; ASSOCIATION;

D O I：

10.1016/j.lpm.2014.07.019

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Angiotensin Converting Enzyme inhibitors (ACE-I) use is a frequent cause of AE and must be suspected systematically in all patients with AE. The risk of AE is increased in: black people, transplanted patients, those who take gliptins or immuhosuppressants (mTOR i). Angiotensin converting enzyme inhibitors induced angioedema (ACE-I AE) can occur a few days to several years after the beginning of this treatment and persist a few months after stopping it. ACE-I AE affect mainly the face and particularly the tongue but also the abdomen. ACE-I AE can be life threatening, due to the involvement of the tongue and the larynx. ACE-I AE must be treated as in the hereditary form when life threatening signs are present, with icatibant or Cl inhibitor concentrate. AE can occur in 10% of angiotensin receptor blockers (ARBs) treated patients who had developed ACE-I AE.

引用

收藏

页码：43 / 47

页数：5

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