Tetrameric complexes of human histocompatibility leukocyte antigen (HLA)-G bind to peripheral blood myelomonocytic cells

被引:206
作者
Allan, DSJ [1 ]
Colonna, M
Lanier, LL
Churakova, TD
Abrams, JS
Ellis, SA
McMichael, AJ
Braud, VM
机构
[1] John Radcliffe Hosp, Inst Mol Med, Oxford OX3 9DS, England
[2] Basel Inst Immunol, CH-4005 Basel, Switzerland
[3] DNAX Res Inst Mol & Cellular Biol Inc, Palo Alto, CA 94304 USA
[4] Inst Anim Hlth, Compton RG20 7NN, England
关键词
immunoglobulin-like transcript; monocyte; natural killer cell; CD94; killer immunoglobulin-like receptor;
D O I
10.1084/jem.189.7.1149
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
The nonclassical MHC class I molecule human histocompatibility leukocyte antigen (HLA)-G is selectively expressed on fetal trophoblast tissue at the maternal-fetal interface in pregnancy. It has long been suggested that HLA-G may inhibit maternal natural killer (NK) cells through interaction with particular NK cell receptors (KIRs). To investigate interactions of HLA-G, we constructed phycoerythrin-labeled tetrameric complexes of HLA-G refolded with a self-peptide. These HLA-G tetramers failed to bind to NK cells and cells transfected with CD94/NKG2 and killer immunoglobulin-like NK receptors. In contrast, HLA-G tetramers did bind to peripheral blood monocytes, staining a CD16(+)CD14(mid) subset with greater intensity. On transfectants, HLA-G tetramers bound to inhibitory immunoglobulin-like transcript (ILT)2 and ILT4 receptors. However, staining in the presence of antibodies reactive with ILT receptors revealed that the interaction of HLA-G tetramers with blood monocytes was largely due to binding to ILT4. These results suggest that the primary role of HLA-G may be the modulation of myelomonocytic cell behavior in pregnancy.
引用
收藏
页码:1149 / 1155
页数:7
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