High efficacy of the BCL-2 inhibitor ABT199 (venetoclax) in BCL-2 high-expressing neuroblastoma cell lines and xenografts and rational for combination with MCL-1 inhibition

被引:42
作者
Bate-Eya, Laurel T. [1 ]
den Hartog, Ilona J. M. [1 ]
van der Ploeg, Ida [1 ]
Schild, Linda [1 ]
Koster, Jan [1 ]
Santo, Evan E. [1 ]
Westerhout, Ellen M. [1 ]
Versteeg, Rogier [1 ]
Caron, Huib N. [2 ]
Molenaar, Jan J. [1 ]
Dolman, M. Emmy M. [1 ]
机构
[1] Univ Amsterdam, Dept Oncogen, Amsterdam, Netherlands
[2] Univ Amsterdam, Acad Med Ctr, Dept Pediat Oncol, Emma Kinderziekenhuis, Meibergdreef 9, NL-1105 AZ Amsterdam, Netherlands
关键词
neuroblastoma; ABT199; BCL-2; MCL-1; resistance; OLIGOMERIZES BAK; LUNG-CANCER; ABT-199; FAMILY; NOXA; RESISTANCE; ABT-737; PROTEIN; DEATH; STABILITY;
D O I
10.18632/oncotarget.8547
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
The anti-apoptotic protein B cell lymphoma/leukaemia 2 (BCL-2) is highly expressed in neuroblastoma and plays an important role in oncogenesis. In this study, the selective BCL-2 inhibitor ABT199 was tested in a panel of neuroblastoma cell lines with diverse expression levels of BCL-2 and other BCL-2 family proteins. ABT199 caused apoptosis more potently in neuroblastoma cell lines expressing high BCL-2 and BIM/BCL-2 complex levels than low expressing cell lines. Effects on cell viability correlated with effects on BIM displacement from BCL-2 and cytochrome c release from the mitochondria. ABT199 treatment of mice with neuroblastoma tumors expressing high BCL-2 levels only resulted in growth inhibition, despite maximum BIM displacement from BCL-2 and the induction of a strong apoptotic response. We showed that neuroblastoma cells might survive ABT199 treatment due to its acute upregulation of the anti-apoptotic BCL-2 family protein myeloid cell leukaemia sequence 1 (MCL-1) and BIM sequestration by MCL-1. In vitro inhibition of MCL-1 sensitized neuroblastoma cell lines to ABT199, confirming the pivotal role of MCL-1 in ABT199 resistance. Our findings suggest that neuroblastoma patients with high BCL-2 and BIM/BCL-2 complex levels might benefit from combination treatment with ABT199 and compounds that inhibit MCL-1 expression.
引用
收藏
页码:27946 / 27958
页数:13
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