Translation deregulation in B-cell lymphomas

被引：5

作者：

Horvilleur, Emilie ^{[1
]}

Wilson, Lindsay A. ^{[1
]}

Willis, Anne E. ^{[1
]}

机构：

[1] Univ Leicester, MRC, Toxicol Unit, Leicester LE1 9HN, Leics, England

来源：

BIOCHEMICAL SOCIETY TRANSACTIONS | 2010年 / 38卷

基金：

英国生物技术与生命科学研究理事会;

关键词：

B cell lymphoma; cancer; immunoglobulin gene rearrangement; internal ribosomal entry segment (IRES); microRNA (miRNA); transcription factor; INTERNAL RIBOSOME ENTRY; C-MYC; MESSENGER-RNA; EXPRESSION; MICRORNAS; RAPAMYCIN; MIR-155; REPRESSION; SURVIVAL; PROFILE;

D O I：

10.1042/BST0381593

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

B cell lymphomas are a heterogeneous group of diseases that can arise at different stages of B cell development often as a result of errors in the cells unique ontogeny Common oncogenic features are often observed including chromosomal rearrangements somatic mutations and transcriptional change Disruption of translation regulation is also frequently implicated in both B cell lymphoma development and progression Deregulation of translation in lymphomagenesis can arise through changes to the proteins constituting the translational machinery or to their regulators and to changes in miRNA (microRNA) expression

引用

页码：1593 / 1597

页数：5

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