Circulating tumour DNA profiling reveals heterogeneity of EGFR inhibitor resistance mechanisms in lung cancer patients (vol 7, 11815, 2016)

被引：30

作者：

Chabon, Jacob J.

Simmons, Andrew D.

Lovejoy, Alexander F.

Esfahani, Mohammad S.

Newman, Aaron M.

Haringsma, Henry J.

Kurtz, David M.

Stehr, Henning

Scherer, Florian

Karlovich, Chris A.

Harding, Thomas C.

Durkin, Kathleen A.

Otterson, Gregory A.

Purcell, W. Thomas

Camidge, D. Ross

Goldman, Jonathan W.

Sequist, Lecia V.

Piotrowska, Zofia

Wakelee, Heather A.

Neal, Joel W.

Alizadeh, Ash A.

Diehn, Maximilian

机构：

来源：

NATURE COMMUNICATIONS | 2016年 / 7卷

关键词：

D O I：

10.1038/ncomms13513

中图分类号：

O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

Nature Communications 7: Article number: 11815 (2016); Published 10 June 2016; Updated 14 November 2016 Previous work by Del Re et al. describing the emergence of KRAS mutations following treatment of non-small cell lung cancer patients with EGFR tyrosine kinase inhibitors was inadvertently omitted from the reference list of this Article and should have been cited as follows.

引用

页数：1

共 1 条

[1] Circulating tumour DNA profiling reveals heterogeneity of EGFR inhibitor resistance mechanisms in lung cancer patients [J].

Chabon, Jacob J. ;

Simmons, Andrew D. ;

Lovejoy, Alexander F. ;

Esfahani, Mohammad S. ;

Newman, Aaron M. ;

Haringsma, Henry J. ;

Kurtz, David M. ;

Stehr, Henning ;

Scherer, Florian ;

Karlovich, Chris A. ;

Harding, Thomas C. ;

Durkin, Kathleen A. ;

Otterson, Gregory A. ;

Purcell, W. Thomas ;

Camidge, D. Ross ;

Goldman, Jonathan W. ;

Sequist, Lecia V. ;

Piotrowska, Zofia ;

Wakelee, Heather A. ;

Neal, Joel W. ;

Alizadeh, Ash A. ;

Diehn, Maximilian .

NATURE COMMUNICATIONS, 2016, 7

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