Response by Caillon et al to Letter Regarding Article, "γδ T Cells Mediate Angiotensin II-Induced Hypertension and Vascular Injury"

被引：1

作者：

Caillon, Antoine ^{[1
]}

Huo, Ku-Geng ^{[1
]}

Paradis, Pierre ^{[1
]}

Schiffrin, Ernesto L. ^{[1
,2
]}

机构：

[1] Sir Mortimer B Davis Jewish Hosp, Lady Davis Inst Med Res, Montreal, PQ, Canada

[2] Sir Mortimer B Davis Jewish Hosp, Dept Med, Montreal, PQ, Canada

来源：

CIRCULATION | 2017年 / 136卷 / 22期

基金：

加拿大健康研究院;

关键词：

D O I：

10.1161/CIRCULATIONAHA.117.030517

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：2200 / 2201

页数：2

共 5 条

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[2] Do intravenous and subcutaneous angiotensin II increase blood pressure by different mechanisms? [J].

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[4] Deficiency of T-regulatory cells exaggerates angiotensin II-induced microvascular injury by enhancing immune responses [J].

Mian, Muhammad Oneeb Rehman ;

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Paradis, Pierre ;

Schiffrin, Ernesto L. .

JOURNAL OF HYPERTENSION, 2016, 34 (01) :97-108

[5] Genetic and Pharmacologic Inhibition of the Chemokine Receptor CXCR2 Prevents Experimental Hypertension and Vascular Dysfunction [J].

Wang, Lei ;

Zhao, Xue-Chen ;

Cui, Wei ;

Ma, Yong-Qiang ;

Ren, Hua-Liang ;

Zhou, Xin ;

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CIRCULATION, 2016, 134 (18) :1353-+

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