Endpoints of Resuscitation

被引：8

作者：

Cestero, Ramon F. ^{[1
]}

Dent, Daniel L. ^{[1
]}

机构：

[1] Univ Texas Hlth Sci Ctr San Antonio, Dept Surg, Div Trauma & Emergency Surg, San Antonio, TX 78229 USA

来源：

SURGICAL CLINICS OF NORTH AMERICA | 2015年 / 95卷 / 02期

关键词：

Shock; Resuscitation; Hypoperfusion; Oxygen demand; GOAL-DIRECTED THERAPY; MECHANICALLY VENTILATED PATIENTS; PREDICTING FLUID RESPONSIVENESS; VENOUS OXYGEN-SATURATION; GASTRIC INTRAMUCOSAL PH; VENA-CAVA DIAMETER; BASE DEFICIT; SUBLINGUAL CAPNOMETRY; SEPTIC SHOCK; RESPIRATORY CHANGES;

D O I：

10.1016/j.suc.2014.10.004

中图分类号：

R61 [外科手术学];

学科分类号：

摘要：

Despite the multiple causes of the shock state, all causes possess the common abnormality of oxygen supply not meeting tissue metabolic demands. Compensatory mechanisms may mask the severity of hypoxemia and hypoperfusion, since catecholamines and extracellular fluid shifts initially compensate for the physiologic derangements associated with patients in shock. Despite the achievement of normal physiologic parameters after resuscitation, significant metabolic acidosis may continue to be present in the tissues, as evidenced by increased lactate levels and metabolic acidosis. This review discusses the major endpoints of resuscitation in clinical use.

引用

页码：319 / +

页数：20

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