A defect of neuronal nitric oxide synthase increases xanthine oxidase derived-superoxide anion and attenuates the control of myocardial oxygen consumption by nitric oxide derived from endothelial nitric oxide synthase

被引：0
|
作者：
Kinugawa, S [1 ]
Huang, HR [1 ]
Wang, ZP [1 ]
Kaminski, PM [1 ]
Wolin, MS [1 ]
Hintze, TH [1 ]
机构：
[1] New York Med Coll, Valhalla, NY 10595 USA
来源：
FASEB JOURNAL | 2005年 / 19卷 / 05期
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D O I：
暂无
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
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页码：A1677 / A1677
页数：1
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