Depression following a spinal cord injury

被引:37
|
作者
Boekamp, JR
Overholser, JC
Schubert, DSP
机构
[1] CASE WESTERN RESERVE UNIV,DEPT PSYCHOL,SCH MED,CLEVELAND,OH 44106
[2] HARVARD UNIV,SCH MED,CAMBRIDGE,MA 02138
关键词
depression; spinal cord injury; social support; coping styles;
D O I
10.2190/CMU6-24AH-E4JG-8KBN
中图分类号
R749 [精神病学];
学科分类号
100205 ;
摘要
Object: Depression is a common problem following a spinal cord injury (SCI) and can greatly interfere with the rehabilitation process because of reduced energy, negative expectations, and social withdrawal. Understanding various factors which influence a vulnerability to depression may improve the diagnosis and treatment of depressive disorders and can improve rehabilitation outcome. Method: A thorough literature search was conducted using Medline, PsychLit, PyschInfo, and Social Science Citation Index to identify relevant articles published between 1967 and 1995. Results: A diathesis-stress model is proposed to explain the increased risk of depressive symptoms after a SCI. Biological changes associated with SCI and pre-existing cognitive biases may influence the individual's vulnerability to stressful life events following the injury. The nature and frequency of stressful life events following the injury can tax the individual's coping resources. Furthermore, the perceived quality of social support and the severity of conflict within the family can influence the individual's adaptation. Conclusions: Social support and recent stressors should be assessed to identify patients at high risk for depression. Patients are less likely to become depressed if their independence is fostered and they are encouraged to develop new sources of self-esteem. Relatives can be counseled to help maintain supportive relationships within the family.
引用
收藏
页码:329 / 349
页数:21
相关论文
共 50 条
  • [41] Bone loss following spinal cord injury
    Otom, Ali H.
    Al-Ahmar, M. Rami
    JOURNAL OF NEURORESTORATOLOGY, 2014, 2 : 81 - 84
  • [42] Colonic neurotransmitters following spinal cord injury
    Lynch A.C.
    Anthony A.
    Dobbs B.
    Frizelle F.A.
    Techniques in Coloproctology, 2000, 4 (2) : 93 - 97
  • [43] Sexual expression following spinal cord injury
    Mona, LR
    Krause, JS
    Norris, FH
    Cameron, RP
    Kalichman, SC
    Lesondak, LM
    NEUROREHABILITATION, 2000, 15 (02) : 121 - 131
  • [44] Anorectal physiology following spinal cord injury
    AC Lynch
    A Anthony
    BR Dobbs
    FA Frizelle
    Spinal Cord, 2000, 38 : 573 - 580
  • [45] Early Spinal Surgery Following Thoracolumbar Spinal Cord Injury
    Agostinello, Jacqui
    Battistuzzo, Camila R.
    Skeers, Peta
    Bernard, Stephen
    Batchelor, Peter E.
    SPINE, 2017, 42 (10) : E617 - E623
  • [46] Spinal circuitry and respiratory recovery following spinal cord injury
    Lane, Michael A.
    Lee, Kun-Ze
    Fuller, David D.
    Reier, Paul J.
    RESPIRATORY PHYSIOLOGY & NEUROBIOLOGY, 2009, 169 (02) : 123 - 132
  • [47] Anorectal physiology following spinal cord injury
    Lynch, AC
    Anthony, A
    Dobbs, BR
    Frizelle, FA
    SPINAL CORD, 2000, 38 (10) : 573 - 580
  • [48] Venlafaxine Extended-Release for Depression Following Spinal Cord Injury A Randomized Clinical Trial
    Fann, Jesse R.
    Bombardier, Charles H.
    Richards, J. Scott
    Wilson, Catherine S.
    Heinemann, Allen W.
    Warren, Ann Marie
    Brooks, Larry
    McCullumsmith, Cheryl B.
    Temkin, Nancy R.
    Warms, Catherine
    Tate, Denise G.
    JAMA PSYCHIATRY, 2015, 72 (03) : 247 - 258
  • [49] Treatment of major depression in individuals with spinal cord injury
    Kemp, BJ
    Kahan, JS
    Krause, JS
    Adkins, RH
    Nava, G
    JOURNAL OF SPINAL CORD MEDICINE, 2004, 27 (01) : 22 - 28
  • [50] Concentrations of glutamate released following spinal cord injury kill oligodendrocytes in the spinal cord
    Xu, GY
    Hughes, MG
    Ye, ZM
    Hulsebosch, CE
    McAdoo, DJ
    EXPERIMENTAL NEUROLOGY, 2004, 187 (02) : 329 - 336