Adaptation to oxidative stress at cellular and tissue level

被引：17

作者：

Akki, Rachid ^{[1
]}

Siracusa, Rosalba ^{[2
]}

Cordaro, Marika ^{[2
]}

Remigante, Alessia ^{[2
]}

Morabito, Rossana ^{[2
]}

Errami, Mohammed ^{[1
]}

Marino, Angela ^{[2
]}

机构：

[1] Univ Abdelmalek Essaadi, Fac Sci, Dept Biol, Tetouan, Morocco

[2] Univ Messina, Dept Chem Biol Pharmaceut & Environm Sci, Viale F Stagno DAlcontres, I-3198166 Messina, Italy

来源：

ARCHIVES OF PHYSIOLOGY AND BIOCHEMISTRY | 2022年 / 128卷 / 02期

关键词：

Preconditioning; ROS; oxidative stress; ischaemia; reperfusion; ISCHEMIA-REPERFUSION INJURY; PEROXIDE-INDUCED APOPTOSIS; OXIDE-INDUCED RESISTANCE; 40; DEGREES-C; HYDROGEN-PEROXIDE; PC12; CELLS; CEREBRAL-ISCHEMIA; NITRIC-OXIDE; GENE-EXPRESSION; PRECONDITIONING PROTECTS;

D O I：

10.1080/13813455.2019.1702059

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Several in vitro and in vivo investigations have already proved that cells and tissues, when pre-exposed to low oxidative stress by different stimuli such as chemical, physical agents and environmental factors, display more resistance against subsequent stronger ischaemic injuries, resulting in an adaptive response known as ischaemic preconditioning (IPC). The aim of this review is to report the most recent knowledge about the complex adaptive mechanisms, including signalling transduction pathways, antioxidant systems, apoptotic and inflammation pathways, underlying cell protection against oxidative damage. In addition, an update about in vivo adaptation strategies in response to ischaemic/reperfusion episodes and brain trauma is also given.

引用

页码：521 / 531

页数：11

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