RELEVANCE OF THE LECTIN PATHWAY OF COMPLEMENT IN RHEUMATIC DISEASES

被引：16

作者：

Boldt, Angelica B. W. ^{[1
]}

Goeldner, Isabela ^{[1
]}

de Messias-Reason, Iara J. T. ^{[1
]}

机构：

[1] Univ Fed Parana, Dept Med Pathol, Mol Immunopathol Lab, BR-80060000 Curitiba, Parana, Brazil

来源：

ADVANCES IN CLINICAL CHEMISTRY, VOL 56 | 2012年 / 56卷

关键词：

MANNOSE-BINDING LECTIN; SYSTEMIC-LUPUS-ERYTHEMATOSUS; PATTERN-RECOGNITION MOLECULES; FICOLIN-3 HAKATA ANTIGEN; PRIMARY SJOGRENS-SYNDROME; GIANT-CELL ARTERITIS; HUMAN ENDOTHELIAL-CELLS; SINGLE STRUCTURAL GENE; INNATE IMMUNE-SYSTEM; C-TYPE LECTIN;

D O I：

10.1016/B978-0-12-394317-0.00012-1

中图分类号：

R446 [实验室诊断]; R-33 [实验医学、医学实验];

学科分类号：

1001 ;

摘要：

Due to its importance both in the clearance of pathogens that contribute as rheumatic etiological agents and in the disposal of apoptotic bodies and potential autoimmune initiators, deficiencies of the components of the lectin pathway of complement have been found to increase susceptibility and modulate the severity of most rheumatic disorders. This chapter introduces the general aspects of the structure, function, and genetics of lectin pathway components and summarizes current knowledge of the field regarding rheumatic diseases predisposition and modulation.

引用

页码：105 / 153

页数：49

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